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Bortezomib reverses a post-translational mechanism of tumorigenesis for patched1 haploinsufficiency in medulloblastoma.
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Bortezomib reverses a post-translational mechanism of tumorigenesis for patched1 haploinsufficiency in medulloblastoma.
Bortezomib reverses a post-translational mechanism of tumorigenesis for patched1 haploinsufficiency in medulloblastoma. Pediatr Blood Cancer. 2009 Aug; 53(2):136-44.
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PubMed
subject areas
Animals
Antineoplastic Agents
Boronic Acids
Bortezomib
Cell Line, Tumor
Cell Transformation, Neoplastic
Cerebellar Neoplasms
Disease Models, Animal
Gene Knock-In Techniques
Hedgehog Proteins
Humans
Immunoblotting
Immunoprecipitation
Loss of Heterozygosity
Medulloblastoma
Mice
Mice, Knockout
Patched Receptors
Patched-1 Receptor
PAX7 Transcription Factor
Proteasome Endopeptidase Complex
Protein Isoforms
Protein Processing, Post-Translational
Pyrazines
Receptors, Cell Surface
authors with profiles
Denis C Guttridge