"Cyclin B1" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A cyclin B subtype that colocalizes with MICROTUBULES during INTERPHASE and is transported into the CELL NUCLEUS at the end of the G2 PHASE.
Descriptor ID |
D056744
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MeSH Number(s) |
D12.644.360.262.120.100 D12.776.167.218.120.100 D12.776.476.262.120.100
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Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Cyclin B1".
Below are MeSH descriptors whose meaning is more specific than "Cyclin B1".
This graph shows the total number of publications written about "Cyclin B1" by people in this website by year, and whether "Cyclin B1" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2000 | 0 | 1 | 1 |
2004 | 0 | 2 | 2 |
2012 | 1 | 0 | 1 |
2013 | 0 | 1 | 1 |
2014 | 1 | 0 | 1 |
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Below are the most recent publications written about "Cyclin B1" by people in Profiles.
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Higher levels of antibodies to the tumour-associated antigen cyclin B1 in cancer-free individuals than in patients with breast cancer. Clin Exp Immunol. 2014 Oct; 178(1):75-8.
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CDK1 stabilizes HIF-1a via direct phosphorylation of Ser668 to promote tumor growth. Cell Cycle. 2013 Dec 01; 12(23):3689-701.
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A genetic variant of Fc?RIIIa is strongly associatedwith humoral immunity to cyclin B1 in African American patients with prostate cancer. Immunogenetics. 2013 Feb; 65(2):91-6.
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Early correction of cell cycle perturbations predicts the immunological response to therapy in HIV-infected patients. AIDS. 2004 Feb 20; 18(3):393-402.
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Distinct capacities of individual E2Fs to induce cell cycle re-entry in postmitotic lens fiber cells of transgenic mice. Dev Neurosci. 2004; 26(5-6):435-45.
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p21Waf1/Cip1/Sdi1-induced growth arrest is associated with depletion of mitosis-control proteins and leads to abnormal mitosis and endoreduplication in recovering cells. Oncogene. 2000 Apr 20; 19(17):2165-70.