Connection

Hongkuan Fan to Lipopolysaccharides

This is a "connection" page, showing publications Hongkuan Fan has written about Lipopolysaccharides.
Connection Strength

2.707
  1. Heterotrimeric Ga(i) proteins are regulated by lipopolysaccharide and are anti-inflammatory in endotoxemia and polymicrobial sepsis. Biochim Biophys Acta. 2011 Mar; 1813(3):466-72.
    View in: PubMed
    Score: 0.323
  2. Lysophosphatidic acid inhibits bacterial endotoxin-induced pro-inflammatory response: potential anti-inflammatory signaling pathways. Mol Med. 2008 Jul-Aug; 14(7-8):422-8.
    View in: PubMed
    Score: 0.271
  3. Differential regulation of lipopolysaccharide and Gram-positive bacteria induced cytokine and chemokine production in macrophages by Galpha(i) proteins. Immunology. 2007 Sep; 122(1):116-23.
    View in: PubMed
    Score: 0.250
  4. Gi proteins regulate lipopolysaccharide and Staphylococcus aureus induced cytokine production but not (1--> 3)-beta-D-glucan induced cytokine suppression. Front Biosci. 2006 Sep 01; 11:2264-74.
    View in: PubMed
    Score: 0.238
  5. Differential regulation of lipopolysaccharide and Gram-positive bacteria induced cytokine and chemokine production in splenocytes by Galphai proteins. Biochim Biophys Acta. 2006 Oct; 1763(10):1051-8.
    View in: PubMed
    Score: 0.237
  6. Lipopolysaccharide- and gram-positive bacteria-induced cellular inflammatory responses: role of heterotrimeric Galpha(i) proteins. Am J Physiol Cell Physiol. 2005 Aug; 289(2):C293-301.
    View in: PubMed
    Score: 0.216
  7. Molecular mechanisms of endotoxin tolerance. J Endotoxin Res. 2004; 10(2):71-84.
    View in: PubMed
    Score: 0.198
  8. Involvement of G(i) proteins and Src tyrosine kinase in TNFalpha production induced by lipopolysaccharide, group B Streptococci and Staphylococcus aureus. Cytokine. 2003 Jun 07; 22(5):126-33.
    View in: PubMed
    Score: 0.191
  9. Exosomes from endothelial progenitor cells improve outcomes of the lipopolysaccharide-induced acute lung injury. Crit Care. 2019 02 13; 23(1):44.
    View in: PubMed
    Score: 0.141
  10. Fli-1 Governs Pericyte Dysfunction in a Murine Model of Sepsis. J Infect Dis. 2018 11 05; 218(12):1995-2005.
    View in: PubMed
    Score: 0.139
  11. Toll-like receptor-induced inflammatory cytokines are suppressed by gain of function or overexpression of Ga(i2) protein. Inflammation. 2012 Oct; 35(5):1611-7.
    View in: PubMed
    Score: 0.091
  12. Beneficial effect of a CXCR4 agonist in murine models of systemic inflammation. Inflammation. 2012 Feb; 35(1):130-7.
    View in: PubMed
    Score: 0.087
  13. Beta-arrestins 1 and 2 differentially regulate LPS-induced signaling and pro-inflammatory gene expression. Mol Immunol. 2007 May; 44(12):3092-9.
    View in: PubMed
    Score: 0.062
  14. The phosphatidylinositol 3 kinase pathway regulates tolerance to lipopolysaccharide and priming responses to Staphylococcus aureus and lipopolysaccharide. Shock. 2006 Jul; 26(1):31-6.
    View in: PubMed
    Score: 0.059
  15. Toll-like receptor 4 coupled GI protein signaling pathways regulate extracellular signal-regulated kinase phosphorylation and AP-1 activation independent of NFkappaB activation. Shock. 2004 Jul; 22(1):57-62.
    View in: PubMed
    Score: 0.051
  16. Differential regulation of cytokine and chemokine production in lipopolysaccharide-induced tolerance and priming. Cytokine. 2004 Jun 07; 26(5):202-8.
    View in: PubMed
    Score: 0.051
  17. Staphylococcus aureus and lipopolysaccharide induce homologous tolerance but heterologous priming: role of interferon-gamma. Shock. 2004 Mar; 21(3):254-60.
    View in: PubMed
    Score: 0.050
  18. Regulation of dendritic cell function improves survival in experimental sepsis through immune chaperone. Innate Immun. 2019 05; 25(4):235-243.
    View in: PubMed
    Score: 0.036
  19. Peroxisome proliferator activated receptor gamma is not necessary for the development of LPS-induced tolerance in macrophages. Immunology. 2008 May; 124(1):51-7.
    View in: PubMed
    Score: 0.016
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.