P. Bell to Cilia
This is a "connection" page, showing publications P. Bell has written about Cilia.
Connection Strength
1.706
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Deletion of airway cilia results in noninflammatory bronchiectasis and hyperreactive airways. Am J Physiol Lung Cell Mol Physiol. 2014 Jan; 306(2):L162-9.
Score: 0.427
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Loss of primary cilia upregulates renal hypertrophic signaling and promotes cystogenesis. J Am Soc Nephrol. 2011 May; 22(5):839-48.
Score: 0.357
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Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells. Am J Physiol Renal Physiol. 2006 Jun; 290(6):F1320-8.
Score: 0.248
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Loss of primary cilia increases polycystin-2 and TRPV4 and the appearance of a nonselective cation channel in the mouse cortical collecting duct. Am J Physiol Renal Physiol. 2019 09 01; 317(3):F632-F637.
Score: 0.158
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Molecular pathways and therapies in autosomal-dominant polycystic kidney disease. Physiology (Bethesda). 2015 May; 30(3):195-207.
Score: 0.118
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TRPP2 and TRPV4 form an EGF-activated calcium permeable channel at the apical membrane of renal collecting duct cells. PLoS One. 2013; 8(8):e73424.
Score: 0.105
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Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors. Am J Physiol Renal Physiol. 2012 Apr 01; 302(7):F801-8.
Score: 0.094
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Cilia movement regulates expression of the Raf-1 kinase inhibitor protein. Am J Physiol Renal Physiol. 2011 May; 300(5):F1163-70.
Score: 0.088
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Heightened epithelial Na+ channel-mediated Na+ absorption in a murine polycystic kidney disease model epithelium lacking apical monocilia. Am J Physiol Cell Physiol. 2006 Apr; 290(4):C952-63.
Score: 0.061
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Epidermal growth factor-induced proliferation of collecting duct cells from Oak Ridge polycystic kidney mice involves activation of Na+/H+ exchanger. Am J Physiol Cell Physiol. 2014 Sep 15; 307(6):C554-60.
Score: 0.028
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The exocyst protein Sec10 interacts with Polycystin-2 and knockdown causes PKD-phenotypes. PLoS Genet. 2011 Apr; 7(4):e1001361.
Score: 0.022