Connection

Yongxia Wu to Graft vs Host Disease

This is a "connection" page, showing publications Yongxia Wu has written about Graft vs Host Disease.
Connection Strength

5.448
  1. MicroRNA-31 regulates T-cell metabolism via HIF1a and promotes chronic GVHD pathogenesis in mice. Blood Adv. 2022 05 24; 6(10):3036-3052.
    View in: PubMed
    Score: 0.692
  2. Donor T-Cell Repertoire Profiling in Recipient Lymphoid and Parenchyma Organs Reveals GVHD Pathogenesis at Clonal Levels After Bone Marrow Transplantation in Mice. Front Immunol. 2021; 12:778996.
    View in: PubMed
    Score: 0.670
  3. STING negatively regulates allogeneic T-cell responses by constraining antigen-presenting cell function. Cell Mol Immunol. 2021 03; 18(3):632-643.
    View in: PubMed
    Score: 0.631
  4. MicroRNA-17-92 is required for T-cell and B-cell pathogenicity in chronic graft-versus-host disease in mice. Blood. 2018 04 26; 131(17):1974-1986.
    View in: PubMed
    Score: 0.517
  5. IL-17A ? Th17 in GvHD. Cell Mol Immunol. 2018 03; 15(3):282-283.
    View in: PubMed
    Score: 0.470
  6. MicroRNA-17-92 controls T-cell responses in graft-versus-host disease and leukemia relapse in mice. Blood. 2015 Sep 10; 126(11):1314-23.
    View in: PubMed
    Score: 0.429
  7. The Absence of IL-12R?2 Expression on Recipient Nonhematopoietic Cells Diminishes Acute Graft-versus-Host Disease in the Gastrointestinal Tract. J Immunol. 2023 02 15; 210(4):486-495.
    View in: PubMed
    Score: 0.182
  8. The druggable transcription factor Fli-1 regulates T cell immunity and tolerance in graft-versus-host disease. J Clin Invest. 2022 11 01; 132(21).
    View in: PubMed
    Score: 0.178
  9. S1P/S1PR1 signaling differentially regulates the allogeneic response of CD4 and CD8 T cells by modulating mitochondrial fission. Cell Mol Immunol. 2022 Nov; 19(11):1235-1250.
    View in: PubMed
    Score: 0.176
  10. Ceramide synthase 6 impacts T-cell allogeneic response and graft-versus-host disease through regulating N-RAS/ERK pathway. Leukemia. 2022 07; 36(7):1907-1915.
    View in: PubMed
    Score: 0.172
  11. XBP-1s Promotes B Cell Pathogenicity in Chronic GVHD by Restraining the Activity of Regulated IRE-1a-Dependent Decay. Front Immunol. 2021; 12:705484.
    View in: PubMed
    Score: 0.165
  12. Interleukin-23 receptor signaling by interleukin-39 potentiates T cell pathogenicity in acute graft-versus-host disease. Am J Transplant. 2021 11; 21(11):3538-3549.
    View in: PubMed
    Score: 0.162
  13. A single strain of Bacteroides fragilis protects gut integrity and reduces GVHD. JCI Insight. 2021 02 08; 6(3).
    View in: PubMed
    Score: 0.158
  14. Targeting the Complement Alternative Pathway Permits Graft Versus Leukemia Activity while Preventing Graft Versus Host Disease. Clin Cancer Res. 2020 07 01; 26(13):3481-3490.
    View in: PubMed
    Score: 0.147
  15. The IL-12 Cytokine and Receptor Family in Graft-vs.-Host Disease. Front Immunol. 2019; 10:988.
    View in: PubMed
    Score: 0.140
  16. Inhibition of the IRE-1a/XBP-1 pathway prevents chronic GVHD and preserves the GVL effect in mice. Blood Adv. 2018 02 27; 2(4):414-427.
    View in: PubMed
    Score: 0.129
  17. T-bet Promotes Acute Graft-versus-Host Disease by Regulating Recipient Hematopoietic Cells in Mice. J Immunol. 2016 Apr 01; 196(7):3168-79.
    View in: PubMed
    Score: 0.112
  18. Inhibition of BTK and ITK with Ibrutinib Is Effective in the Prevention of Chronic Graft-versus-Host Disease in Mice. PLoS One. 2015; 10(9):e0137641.
    View in: PubMed
    Score: 0.109
  19. Essential Role of Interleukin-12/23p40 in the Development of Graft-versus-Host Disease in Mice. Biol Blood Marrow Transplant. 2015 Jul; 21(7):1195-204.
    View in: PubMed
    Score: 0.105
  20. T-bet is critical for the development of acute graft-versus-host disease through controlling T cell differentiation and function. J Immunol. 2015 Jan 01; 194(1):388-97.
    View in: PubMed
    Score: 0.103
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.