Connection

Hongkuan Fan to Mice

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This is a "connection" page, showing publications Hongkuan Fan has written about Mice.
Hongkuan Fan
Connection Strength
1.232
Mice
  1. lncRNA Neat1 regulates neuronal dysfunction post-sepsis via stabilization of hemoglobin subunit beta. Mol Ther. 2022 07 06; 30(7):2618-2632.
    View in: PubMed
    Score: 0.088
  2. Suppression of Fli-1 protects against pericyte loss and cognitive deficits in Alzheimer's disease. Mol Ther. 2022 04 06; 30(4):1451-1464.
    View in: PubMed
    Score: 0.087
  3. miR-145a Regulation of Pericyte Dysfunction in a Murine Model of Sepsis. J Infect Dis. 2020 08 17; 222(6):1037-1045.
    View in: PubMed
    Score: 0.079
  4. Exosomes from endothelial progenitor cells improve outcomes of the lipopolysaccharide-induced acute lung injury. Crit Care. 2019 02 13; 23(1):44.
    View in: PubMed
    Score: 0.071
  5. Fli-1 transcription factor regulates the expression of caspase-1 in lung pericytes. Mol Immunol. 2019 04; 108:1-7.
    View in: PubMed
    Score: 0.071
  6. Application of Deacetylated Poly-N-Acetyl Glucosamine Nanoparticles for the Delivery of miR-126 for the Treatment of Cecal Ligation and Puncture-Induced Sepsis. Inflammation. 2019 Feb; 42(1):170-184.
    View in: PubMed
    Score: 0.071
  7. Fli-1 Governs Pericyte Dysfunction in a Murine Model of Sepsis. J Infect Dis. 2018 11 05; 218(12):1995-2005.
    View in: PubMed
    Score: 0.069
  8. Exosomes from Endothelial Progenitor Cells Improve the Outcome of a Murine Model of Sepsis. Mol Ther. 2018 05 02; 26(5):1375-1384.
    View in: PubMed
    Score: 0.066
  9. The role of miRNAs in cardiovascular disease risk factors. Atherosclerosis. 2016 11; 254:271-281.
    View in: PubMed
    Score: 0.060
  10. Endothelial progenitor cells and a stromal cell-derived factor-1a analogue synergistically improve survival in sepsis. Am J Respir Crit Care Med. 2014 Jun 15; 189(12):1509-19.
    View in: PubMed
    Score: 0.051
  11. Toll-like receptor-induced inflammatory cytokines are suppressed by gain of function or overexpression of Ga(i2) protein. Inflammation. 2012 Oct; 35(5):1611-7.
    View in: PubMed
    Score: 0.046
  12. Beneficial effect of a CXCR4 agonist in murine models of systemic inflammation. Inflammation. 2012 Feb; 35(1):130-7.
    View in: PubMed
    Score: 0.043
  13. Increased expression of beta-arrestin 1 and 2 in murine models of rheumatoid arthritis: isoform specific regulation of inflammation. Mol Immunol. 2011 Oct; 49(1-2):64-74.
    View in: PubMed
    Score: 0.042
  14. Heterotrimeric Ga(i) proteins are regulated by lipopolysaccharide and are anti-inflammatory in endotoxemia and polymicrobial sepsis. Biochim Biophys Acta. 2011 Mar; 1813(3):466-72.
    View in: PubMed
    Score: 0.040
  15. Beta-arrestin 2 negatively regulates sepsis-induced inflammation. Immunology. 2010 Jul; 130(3):344-51.
    View in: PubMed
    Score: 0.039
  16. Lysophosphatidic acid inhibits bacterial endotoxin-induced pro-inflammatory response: potential anti-inflammatory signaling pathways. Mol Med. 2008 Jul-Aug; 14(7-8):422-8.
    View in: PubMed
    Score: 0.034
  17. Differential regulation of lipopolysaccharide and Gram-positive bacteria induced cytokine and chemokine production in macrophages by Galpha(i) proteins. Immunology. 2007 Sep; 122(1):116-23.
    View in: PubMed
    Score: 0.031
  18. Beta-arrestins 1 and 2 differentially regulate LPS-induced signaling and pro-inflammatory gene expression. Mol Immunol. 2007 May; 44(12):3092-9.
    View in: PubMed
    Score: 0.031
  19. Differential regulation of lipopolysaccharide and Gram-positive bacteria induced cytokine and chemokine production in splenocytes by Galphai proteins. Biochim Biophys Acta. 2006 Oct; 1763(10):1051-8.
    View in: PubMed
    Score: 0.030
  20. Lipopolysaccharide- and gram-positive bacteria-induced cellular inflammatory responses: role of heterotrimeric Galpha(i) proteins. Am J Physiol Cell Physiol. 2005 Aug; 289(2):C293-301.
    View in: PubMed
    Score: 0.027
  21. Involvement of G(i) proteins and Src tyrosine kinase in TNFalpha production induced by lipopolysaccharide, group B Streptococci and Staphylococcus aureus. Cytokine. 2003 Jun 07; 22(5):126-33.
    View in: PubMed
    Score: 0.024
  22. Selection of peptide ligands binding to fibroblast growth factor receptor 1. IUBMB Life. 2002 Aug; 54(2):67-72.
    View in: PubMed
    Score: 0.023
  23. Sectm1a Facilitates Protection against Inflammation-Induced Organ Damage through Promoting TRM Self-Renewal. Mol Ther. 2021 03 03; 29(3):1294-1311.
    View in: PubMed
    Score: 0.020
  24. Expression of GM-CSF Is Regulated by Fli-1 Transcription Factor, a Potential Drug Target. J Immunol. 2021 01 01; 206(1):59-66.
    View in: PubMed
    Score: 0.020
  25. Regulation of dendritic cell function improves survival in experimental sepsis through immune chaperone. Innate Immun. 2019 05; 25(4):235-243.
    View in: PubMed
    Score: 0.018
  26. Kallistatin treatment attenuates lethality and organ injury in mouse models of established sepsis. Crit Care. 2015 May 01; 19:200.
    View in: PubMed
    Score: 0.014
  27. Human kallistatin administration reduces organ injury and improves survival in a mouse model of polymicrobial sepsis. Immunology. 2014 Jun; 142(2):216-26.
    View in: PubMed
    Score: 0.013
  28. Peroxisome proliferator-activated receptor {delta} regulates inflammation via NF-{kappa}B signaling in polymicrobial sepsis. Am J Pathol. 2010 Oct; 177(4):1834-47.
    View in: PubMed
    Score: 0.010
  29. Peroxisome proliferator activated receptor gamma is not necessary for the development of LPS-induced tolerance in macrophages. Immunology. 2008 May; 124(1):51-7.
    View in: PubMed
    Score: 0.008
  30. The phosphatidylinositol 3 kinase pathway regulates tolerance to lipopolysaccharide and priming responses to Staphylococcus aureus and lipopolysaccharide. Shock. 2006 Jul; 26(1):31-6.
    View in: PubMed
    Score: 0.007
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.