Connection

Mark Kindy to Disease Models, Animal

This is a "connection" page, showing publications Mark Kindy has written about Disease Models, Animal.
Connection Strength

1.805
  1. High-Sodium Diet Has Opposing Effects on?Mean Arterial Blood Pressure and?Cerebral Perfusion in a Transgenic Mouse Model of Alzheimer's Disease. J Alzheimers Dis. 2016 10 04; 54(3):1061-1072.
    View in: PubMed
    Score: 0.314
  2. Dietary supplementation of GrandFusion(?) mitigates cerebral ischemia-induced neuronal damage and attenuates inflammation. Nutr Neurosci. 2016 Sep; 19(7):290-300.
    View in: PubMed
    Score: 0.283
  3. A contusion model of severe spinal cord injury in rats. J Vis Exp. 2013 Aug 17; (78).
    View in: PubMed
    Score: 0.253
  4. Plant-Based Nutritional Supplementation Attenuates LPS-Induced Low-Grade Systemic Activation. Int J Mol Sci. 2021 Jan 08; 22(2).
    View in: PubMed
    Score: 0.105
  5. Effects of GrandFusion Diet on Cognitive Impairment in Transgenic Mouse Model of Alzheimer's Disease. Nutrients. 2020 Dec 30; 13(1).
    View in: PubMed
    Score: 0.105
  6. Impact of nutrition on inflammation, tauopathy, and behavioral outcomes from chronic traumatic encephalopathy. J Neuroinflammation. 2018 Sep 24; 15(1):277.
    View in: PubMed
    Score: 0.090
  7. The cysteine protease cathepsin B is a key drug target and cysteine protease inhibitors are potential therapeutics for traumatic brain injury. J Neurotrauma. 2014 Mar 01; 31(5):515-29.
    View in: PubMed
    Score: 0.065
  8. Deletion of the cathepsin B gene improves memory deficits in a transgenic ALZHeimer's disease mouse model expressing A?PP containing the wild-type ?-secretase site sequence. J Alzheimers Dis. 2012; 29(4):827-40.
    View in: PubMed
    Score: 0.056
  9. Vitamin D3-enriched diet correlates with a decrease of amyloid plaques in the brain of A?PP transgenic mice. J Alzheimers Dis. 2011; 25(2):295-307.
    View in: PubMed
    Score: 0.053
  10. The cysteine protease inhibitor, E64d, reduces brain amyloid-? and improves memory deficits in Alzheimer's disease animal models by inhibiting cathepsin B, but not BACE1, ?-secretase activity. J Alzheimers Dis. 2011; 26(2):387-408.
    View in: PubMed
    Score: 0.053
  11. Pharmacogenetic features of cathepsin B inhibitors that improve memory deficit and reduce beta-amyloid related to Alzheimer's disease. Biol Chem. 2010 Aug; 391(8):861-72.
    View in: PubMed
    Score: 0.051
  12. The dual role of tumor necrosis factor-alpha in the pathophysiology of spinal cord injury. Neurosci Lett. 2008 Jun 20; 438(2):174-9.
    View in: PubMed
    Score: 0.044
  13. Delta 2-specific opioid receptor agonist and hibernating woodchuck plasma fraction provide ischemic neuroprotection. Acad Emerg Med. 2008 Mar; 15(3):250-7.
    View in: PubMed
    Score: 0.043
  14. Cholesterol and Alzheimer's disease: clinical and experimental models suggest interactions of different genetic, dietary and environmental risk factors. Curr Drug Targets. 2004 Aug; 5(6):517-28.
    View in: PubMed
    Score: 0.034
  15. Fibroblast growth factor-18 reduced infarct volumes and behavioral deficits after transient occlusion of the middle cerebral artery in rats. Stroke. 2003 Jun; 34(6):1507-12.
    View in: PubMed
    Score: 0.031
  16. Inflammation-dependent cerebral deposition of serum amyloid a protein in a mouse model of amyloidosis. J Neurosci. 2002 Jul 15; 22(14):5900-9.
    View in: PubMed
    Score: 0.029
  17. Cathepsin B Gene Knockout Improves Behavioral Deficits and Reduces Pathology in Models of Neurologic Disorders. Pharmacol Rev. 2022 07; 74(3):600-629.
    View in: PubMed
    Score: 0.029
  18. Exogenous Ketones Lower Blood Glucose Level in Rested and Exercised Rodent Models. Nutrients. 2019 Oct 01; 11(10).
    View in: PubMed
    Score: 0.024
  19. Reduction in amyloid A amyloid formation in apolipoprotein-E-deficient mice. Am J Pathol. 1998 May; 152(5):1387-95.
    View in: PubMed
    Score: 0.022
  20. Association of apolipoprotein E with murine amyloid A protein amyloid. Lab Invest. 1995 Oct; 73(4):469-75.
    View in: PubMed
    Score: 0.018
  21. Biomaterial-based interventions for neuronal regeneration and functional recovery in rodent model of spinal cord injury: a systematic review. J Spinal Cord Med. 2013 May; 36(3):174-90.
    View in: PubMed
    Score: 0.015
  22. GLP-1 receptor stimulation preserves primary cortical and dopaminergic neurons in cellular and rodent models of stroke and Parkinsonism. Proc Natl Acad Sci U S A. 2009 Jan 27; 106(4):1285-90.
    View in: PubMed
    Score: 0.011
  23. Focal cerebral ischemia in the TNFalpha-transgenic rat. J Neuroinflammation. 2008 Oct 23; 5:47.
    View in: PubMed
    Score: 0.011
  24. High cholesterol-induced neuroinflammation and amyloid precursor protein processing correlate with loss of working memory in mice. J Neurochem. 2008 Jul; 106(1):475-85.
    View in: PubMed
    Score: 0.011
  25. Inhibitors of cathepsin B improve memory and reduce beta-amyloid in transgenic Alzheimer disease mice expressing the wild-type, but not the Swedish mutant, beta-secretase site of the amyloid precursor protein. J Biol Chem. 2008 Mar 21; 283(12):7745-53.
    View in: PubMed
    Score: 0.011
  26. Estradiol enhances neurogenesis following ischemic stroke through estrogen receptors alpha and beta. J Comp Neurol. 2007 Feb 20; 500(6):1064-75.
    View in: PubMed
    Score: 0.010
  27. RAGE potentiates Abeta-induced perturbation of neuronal function in transgenic mice. EMBO J. 2004 Oct 13; 23(20):4096-105.
    View in: PubMed
    Score: 0.009
  28. Neprilysin regulates amyloid Beta peptide levels. J Mol Neurosci. 2004; 22(1-2):5-11.
    View in: PubMed
    Score: 0.008
  29. Neprilysin gene transfer reduces human amyloid pathology in transgenic mice. J Neurosci. 2003 Mar 15; 23(6):1992-6.
    View in: PubMed
    Score: 0.008
  30. Abeta deposition is essential to AD neuropathology. J Alzheimers Dis. 2002 Jun; 4(3):133-8.
    View in: PubMed
    Score: 0.007
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.