Connection

Louis Luttrell to Mice

This is a "connection" page, showing publications Louis Luttrell has written about Mice.
Connection Strength

0.515
  1. Transcriptomic characterization of signaling pathways associated with osteoblastic differentiation of MC-3T3E1 cells. PLoS One. 2019; 14(1):e0204197.
    View in: PubMed
    Score: 0.067
  2. Informatic deconvolution of biased GPCR signaling mechanisms from in vivo pharmacological experimentation. Methods. 2016 Jan 01; 92:51-63.
    View in: PubMed
    Score: 0.052
  3. Emergent biological properties of arrestin pathway-selective biased agonism. J Recept Signal Transduct Res. 2013 Jun; 33(3):153-61.
    View in: PubMed
    Score: 0.045
  4. Biasing the parathyroid hormone receptor: relating in vitro ligand efficacy to in vivo biological activity. Methods Enzymol. 2013; 522:229-62.
    View in: PubMed
    Score: 0.044
  5. A beta-arrestin-biased agonist of the parathyroid hormone receptor (PTH1R) promotes bone formation independent of G protein activation. Sci Transl Med. 2009 Oct 07; 1(1):1ra1.
    View in: PubMed
    Score: 0.035
  6. Insulin-like growth factor-2/mannose-6 phosphate receptors. Vitam Horm. 2009; 80:667-97.
    View in: PubMed
    Score: 0.034
  7. beta-Arrestin 2 expression determines the transcriptional response to lysophosphatidic acid stimulation in murine embryo fibroblasts. J Biol Chem. 2005 Sep 16; 280(37):32157-67.
    View in: PubMed
    Score: 0.026
  8. Ectodomain shedding-dependent transactivation of epidermal growth factor receptors in response to insulin-like growth factor type I. Mol Endocrinol. 2004 Nov; 18(11):2727-39.
    View in: PubMed
    Score: 0.025
  9. Transactivation of the epidermal growth factor receptor mediates parathyroid hormone and prostaglandin F2 alpha-stimulated mitogen-activated protein kinase activation in cultured transgenic murine osteoblasts. Mol Endocrinol. 2003 Aug; 17(8):1607-21.
    View in: PubMed
    Score: 0.023
  10. ?-Arrestin2 is a critical component of the GPCR-eNOS signalosome. Proc Natl Acad Sci U S A. 2020 05 26; 117(21):11483-11492.
    View in: PubMed
    Score: 0.018
  11. Exploring G protein-coupled receptor signaling networks using SILAC-based phosphoproteomics. Methods. 2016 Jan 01; 92:36-50.
    View in: PubMed
    Score: 0.013
  12. Epidermal growth factor-induced proliferation of collecting duct cells from Oak Ridge polycystic kidney mice involves activation of Na+/H+ exchanger. Am J Physiol Cell Physiol. 2014 Sep 15; 307(6):C554-60.
    View in: PubMed
    Score: 0.012
  13. ?-arrestin-selective G protein-coupled receptor agonists engender unique biological efficacy in vivo. Mol Endocrinol. 2013 Feb; 27(2):296-314.
    View in: PubMed
    Score: 0.011
  14. Increased expression of beta-arrestin 1 and 2 in murine models of rheumatoid arthritis: isoform specific regulation of inflammation. Mol Immunol. 2011 Oct; 49(1-2):64-74.
    View in: PubMed
    Score: 0.010
  15. A pertussis toxin-sensitive G-protein mediates some aspects of insulin action in BC3H-1 murine myocytes. J Biol Chem. 1990 Oct 05; 265(28):16873-9.
    View in: PubMed
    Score: 0.009
  16. Bradykinin decreases podocyte permeability through ADAM17-dependent epidermal growth factor receptor activation and zonula occludens-1 rearrangement. J Pharmacol Exp Ther. 2010 Sep 01; 334(3):775-83.
    View in: PubMed
    Score: 0.009
  17. HDL3, but not HDL2, stimulates plasminogen activator inhibitor-1 release from adipocytes: the role of sphingosine-1-phosphate. J Lipid Res. 2010 Sep; 51(9):2619-28.
    View in: PubMed
    Score: 0.009
  18. Beta-arrestin 2 negatively regulates sepsis-induced inflammation. Immunology. 2010 Jul; 130(3):344-51.
    View in: PubMed
    Score: 0.009
  19. Insulin and phorbol ester induce distinct phosphorylations of pp60c-src in the BC3H-1 murine myocyte cell line. Oncogene. 1989 Mar; 4(3):317-24.
    View in: PubMed
    Score: 0.008
  20. Beta-arrestins 1 and 2 differentially regulate LPS-induced signaling and pro-inflammatory gene expression. Mol Immunol. 2007 May; 44(12):3092-9.
    View in: PubMed
    Score: 0.007
  21. Hexose-independent activation of glycogen synthase and pyruvate dehydrogenase by insulin is dissociated in the mouse BC3H-1 cell line. Mol Pharmacol. 1986 Dec; 30(6):624-30.
    View in: PubMed
    Score: 0.007
  22. Beta-arrestin- and G protein receptor kinase-mediated calcium-sensing receptor desensitization. Mol Endocrinol. 2005 Apr; 19(4):1078-87.
    View in: PubMed
    Score: 0.006
  23. Dual inhibition of beta-adrenergic and angiotensin II receptors by a single antagonist: a functional role for receptor-receptor interaction in vivo. Circulation. 2003 Sep 30; 108(13):1611-8.
    View in: PubMed
    Score: 0.006
  24. Protein kinase A and G protein-coupled receptor kinase phosphorylation mediates beta-1 adrenergic receptor endocytosis through different pathways. J Biol Chem. 2003 Sep 12; 278(37):35403-11.
    View in: PubMed
    Score: 0.006
  25. beta -Arrestin-mediated recruitment of the Src family kinase Yes mediates endothelin-1-stimulated glucose transport. J Biol Chem. 2001 Nov 23; 276(47):43663-7.
    View in: PubMed
    Score: 0.005
  26. Direct binding of activated c-Src to the beta 3-adrenergic receptor is required for MAP kinase activation. J Biol Chem. 2000 Dec 08; 275(49):38131-4.
    View in: PubMed
    Score: 0.005
  27. The beta3-adrenergic receptor activates mitogen-activated protein kinase in adipocytes through a Gi-dependent mechanism. J Biol Chem. 1999 Apr 23; 274(17):12017-22.
    View in: PubMed
    Score: 0.004
  28. Targeting the receptor-Gq interface to inhibit in vivo pressure overload myocardial hypertrophy. Science. 1998 Apr 24; 280(5363):574-7.
    View in: PubMed
    Score: 0.004
  29. Augmented mitogenic responsiveness to epidermal growth factor in murine fibroblasts that overexpress pp60c-src. Mol Cell Biol. 1988 Jan; 8(1):497-501.
    View in: PubMed
    Score: 0.002
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.