Connection

Deepak Nihalani to Mice

This is a "connection" page, showing publications Deepak Nihalani has written about Mice.
Connection Strength

0.594
  1. Phosphorylation of slit diaphragm proteins NEPHRIN and NEPH1 upon binding of HGF promotes podocyte repair. J Biol Chem. 2021 09; 297(3):101079.
    View in: PubMed
    Score: 0.082
  2. Disruption of the exocyst induces podocyte loss and dysfunction. J Biol Chem. 2019 06 28; 294(26):10104-10119.
    View in: PubMed
    Score: 0.070
  3. Mitochondrial biogenesis induced by the ?2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury. Kidney Int. 2019 09; 96(3):656-673.
    View in: PubMed
    Score: 0.070
  4. The motor protein Myo1c regulates transforming growth factor-?-signaling and fibrosis in podocytes. Kidney Int. 2019 07; 96(1):139-158.
    View in: PubMed
    Score: 0.069
  5. Targeting Neph1 and ZO-1 protein-protein interaction in podocytes prevents podocyte injury and preserves glomerular filtration function. Sci Rep. 2017 09 21; 7(1):12047.
    View in: PubMed
    Score: 0.062
  6. Myo1c is an unconventional myosin required for zebrafish glomerular development. Kidney Int. 2013 Dec; 84(6):1154-65.
    View in: PubMed
    Score: 0.046
  7. Src family kinases directly regulate JIP1 module dynamics and activation. Mol Cell Biol. 2007 Apr; 27(7):2431-41.
    View in: PubMed
    Score: 0.030
  8. Loss of Motor Protein MYO1C Causes Rhodopsin Mislocalization and Results in Impaired Visual Function. Cells. 2021 05 26; 10(6).
    View in: PubMed
    Score: 0.020
  9. Targeting myosin 1c inhibits murine hepatic fibrogenesis. Am J Physiol Gastrointest Liver Physiol. 2021 06 01; 320(6):G1044-G1053.
    View in: PubMed
    Score: 0.020
  10. The Use of High-Throughput Transcriptomics to Identify Pathways with Therapeutic Significance in Podocytes. Int J Mol Sci. 2019 Dec 31; 21(1).
    View in: PubMed
    Score: 0.018
  11. High-content screening assay-based discovery of paullones as novel podocyte-protective agents. Am J Physiol Renal Physiol. 2018 02 01; 314(2):F280-F292.
    View in: PubMed
    Score: 0.016
  12. The exocyst is required for photoreceptor ciliogenesis and retinal development. J Biol Chem. 2017 09 08; 292(36):14814-14826.
    View in: PubMed
    Score: 0.015
  13. Deficiency of the Angiotensinase Aminopeptidase A Increases Susceptibility to Glomerular Injury. J Am Soc Nephrol. 2017 Jul; 28(7):2119-2132.
    View in: PubMed
    Score: 0.015
  14. A reassessment of soluble urokinase-type plasminogen activator receptor in glomerular disease. Kidney Int. 2015 Mar; 87(3):564-74.
    View in: PubMed
    Score: 0.013
  15. Functional study of mammalian Neph proteins in Drosophila melanogaster. PLoS One. 2012; 7(7):e40300.
    View in: PubMed
    Score: 0.011
  16. Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease. J Clin Invest. 2012 Feb; 122(2):674-92.
    View in: PubMed
    Score: 0.011
  17. Neph1 cooperates with nephrin to transduce a signal that induces actin polymerization. Mol Cell Biol. 2007 Dec; 27(24):8698-712.
    View in: PubMed
    Score: 0.008
  18. Nephrin ectodomain engagement results in Src kinase activation, nephrin phosphorylation, Nck recruitment, and actin polymerization. J Clin Invest. 2006 May; 116(5):1346-59.
    View in: PubMed
    Score: 0.007
  19. Inhibition of membrane depolarisation-induced transcriptional activity of cyclic AMP response element binding protein (CREB) by the dual-leucine-zipper-bearing kinase in a pancreatic islet beta cell line. Diabetologia. 2006 Feb; 49(2):332-42.
    View in: PubMed
    Score: 0.007
  20. Fyn binds to and phosphorylates the kidney slit diaphragm component Nephrin. J Biol Chem. 2003 Jun 06; 278(23):20716-23.
    View in: PubMed
    Score: 0.006
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.