Co-Authors
This is a "connection" page, showing publications co-authored by Deepak Nihalani and Ehtesham Arif.
Connection Strength
6.152
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Mitochondrial biogenesis induced by the ?2-adrenergic receptor agonist formoterol accelerates podocyte recovery from glomerular injury. Kidney Int. 2019 09; 96(3):656-673.
Score: 0.706
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Beta2-adrenergic receptor in kidney biology: A current prospective. Nephrology (Carlton). 2019 May; 24(5):497-503.
Score: 0.706
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The motor protein Myo1c regulates transforming growth factor-?-signaling and fibrosis in podocytes. Kidney Int. 2019 07; 96(1):139-158.
Score: 0.698
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Structural Analysis of the Myo1c and Neph1 Complex Provides Insight into the Intracellular Movement of Neph1. Mol Cell Biol. 2016 06 01; 36(11):1639-54.
Score: 0.575
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Adriamycin susceptibility among C57BL/6 substrains. Kidney Int. 2016 Mar; 89(3):721-3.
Score: 0.567
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Slit diaphragm protein Neph1 and its signaling: a novel therapeutic target for protection of podocytes against glomerular injury. J Biol Chem. 2014 Apr 04; 289(14):9502-18.
Score: 0.492
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Glomerular Filtration Barrier Assembly: An insight. Postdoc J. 2013 Apr; 1(4):33-45.
Score: 0.463
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Phosphorylation of slit diaphragm proteins NEPHRIN and NEPH1 upon binding of HGF promotes podocyte repair. J Biol Chem. 2021 09; 297(3):101079.
Score: 0.207
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Targeting myosin 1c inhibits murine hepatic fibrogenesis. Am J Physiol Gastrointest Liver Physiol. 2021 06 01; 320(6):G1044-G1053.
Score: 0.203
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Transcriptomics Reveal Altered Metabolic and Signaling Pathways in Podocytes Exposed to C16 Ceramide-Enriched Lipoproteins. Genes (Basel). 2020 02 07; 11(2).
Score: 0.186
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The Use of High-Throughput Transcriptomics to Identify Pathways with Therapeutic Significance in Podocytes. Int J Mol Sci. 2019 Dec 31; 21(1).
Score: 0.185
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Mutations in KIRREL1, a slit diaphragm component, cause steroid-resistant nephrotic syndrome. Kidney Int. 2019 10; 96(4):883-889.
Score: 0.179
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Disruption of the exocyst induces podocyte loss and dysfunction. J Biol Chem. 2019 06 28; 294(26):10104-10119.
Score: 0.177
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Development of a novel cell-based assay to diagnose recurrent focal segmental glomerulosclerosis patients. Kidney Int. 2019 03; 95(3):708-716.
Score: 0.173
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Targeting Neph1 and ZO-1 protein-protein interaction in podocytes prevents podocyte injury and preserves glomerular filtration function. Sci Rep. 2017 09 21; 7(1):12047.
Score: 0.158
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Myo1c is an unconventional myosin required for zebrafish glomerular development. Kidney Int. 2013 Dec; 84(6):1154-65.
Score: 0.117
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Solution structure analysis of cytoplasmic domain of podocyte protein Neph1 using small/wide angle x-ray scattering (SWAXS). J Biol Chem. 2012 Mar 16; 287(12):9441-53.
Score: 0.106
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Ischemic injury to kidney induces glomerular podocyte effacement and dissociation of slit diaphragm proteins Neph1 and ZO-1. J Biol Chem. 2008 Dec 19; 283(51):35579-89.
Score: 0.085
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Loss of Motor Protein MYO1C Causes Rhodopsin Mislocalization and Results in Impaired Visual Function. Cells. 2021 05 26; 10(6).
Score: 0.051
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A Novel CLCN5 Mutation Associated With?Focal Segmental Glomerulosclerosis and?Podocyte Injury. Kidney Int Rep. 2018 Nov; 3(6):1443-1453.
Score: 0.042
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High-content screening assay-based discovery of paullones as novel podocyte-protective agents. Am J Physiol Renal Physiol. 2018 02 01; 314(2):F280-F292.
Score: 0.040
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Deficiency of the Angiotensinase Aminopeptidase A Increases Susceptibility to Glomerular Injury. J Am Soc Nephrol. 2017 Jul; 28(7):2119-2132.
Score: 0.038