Edward Hogan to Rats, Sprague-Dawley
This is a "connection" page, showing publications Edward Hogan has written about Rats, Sprague-Dawley.
Connection Strength
0.306
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Treatment of spinal cord impact injury in the rat with transforming growth factor-beta. J Neurol Sci. 2002 Aug 15; 200(1-2):33-41.
Score: 0.035
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Inhibition of proteolysis by a cyclooxygenase inhibitor, indomethacin. Neurochem Res. 2000 Nov; 25(11):1509-15.
Score: 0.031
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Immunohistochemical localization of kininogen in rat spinal cord and brain. Exp Neurol. 1999 Oct; 159(2):528-37.
Score: 0.029
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Extracellular signal-regulated kinase and p38 subgroups of mitogen-activated protein kinases regulate inducible nitric oxide synthase and tumor necrosis factor-alpha gene expression in endotoxin-stimulated primary glial cultures. J Neurosci. 1998 Mar 01; 18(5):1633-41.
Score: 0.026
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Increased calpain content and progressive degradation of neurofilament protein in spinal cord injury. Brain Res. 1997 Mar 28; 752(1-2):301-6.
Score: 0.024
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A new mechanism of methylprednisolone and other corticosteroids action demonstrated in vitro: inhibition of a proteinase (calpain) prevents myelin and cytoskeletal protein degradation. Brain Res. 1997 Feb 14; 748(1-2):205-10.
Score: 0.024
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Neuron-microglia interaction induced bi-directional cytotoxicity associated with calpain activation. J Neurochem. 2016 11; 139(3):440-455.
Score: 0.024
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Calcium-activated neutral proteinase (calpain) in rat brain during development: compartmentation and role in myelination. Brain Res Dev Brain Res. 1993 Jan 15; 71(1):107-13.
Score: 0.018
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Higher calpastatin levels correlate with resistance to calpain-mediated proteolysis and neuronal apoptosis in juvenile rats after spinal cord injury. J Neurotrauma. 2004 Sep; 21(9):1240-54.
Score: 0.010
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Relatively low levels of calpain expression in juvenile rat correlate with less neuronal apoptosis after spinal cord injury. Exp Neurol. 2004 Jun; 187(2):529-32.
Score: 0.010
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Early induction of secondary injury factors causing activation of calpain and mitochondria-mediated neuronal apoptosis following spinal cord injury in rats. J Neurosci Res. 2003 Jul 01; 73(1):95-104.
Score: 0.009
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Cell death in spinal cord injury (SCI) requires de novo protein synthesis. Calpain inhibitor E-64-d provides neuroprotection in SCI lesion and penumbra. Ann N Y Acad Sci. 2001 Jun; 939:436-49.
Score: 0.008
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Increased calpain expression is associated with apoptosis in rat spinal cord injury: calpain inhibitor provides neuroprotection. Neurochem Res. 2000 Oct; 25(9-10):1191-8.
Score: 0.008
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Calpain activity and expression increased in activated glial and inflammatory cells in penumbra of spinal cord injury lesion. J Neurosci Res. 2000 Jul 15; 61(2):146-50.
Score: 0.008
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E-64-d prevents both calpain upregulation and apoptosis in the lesion and penumbra following spinal cord injury in rats. Brain Res. 2000 Jun 09; 867(1-2):80-9.
Score: 0.008
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Calpain activity and translational expression increased in spinal cord injury. Brain Res. 1999 Jan 23; 816(2):375-80.
Score: 0.007
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New inhibitors of calpain prevent degradation of cytoskeletal and myelin proteins in spinal cord in vitro. J Neurosci Res. 1998 Jan 15; 51(2):218-22.
Score: 0.006
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Role of calpain in spinal cord injury: increased calpain immunoreactivity in rat spinal cord after impact trauma. Neurochem Res. 1996 Apr; 21(4):441-8.
Score: 0.006
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Role of calpain in spinal cord injury: increased mcalpain immunoreactivity in spinal cord after compression injury in the rat. Neurochem Int. 1995 Oct-Nov; 27(4-5):425-32.
Score: 0.005
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Effect of U-50488h, a selective opioid kappa receptor agonist, on vascular injury after spinal cord trauma. Brain Res. 1993 Oct 29; 626(1-2):45-9.
Score: 0.005
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Protective effect of methylprednisolone on vascular injury in rat spinal cord injury. J Neurotrauma. 1992; 9(3):245-53.
Score: 0.004