Connection

David Jollow to Acetaminophen

This is a "connection" page, showing publications David Jollow has written about Acetaminophen.
Connection Strength

1.299
  1. Modulation of macrophage functioning abrogates the acute hepatotoxicity of acetaminophen. Hepatology. 1995 Apr; 21(4):1045-50.
    View in: PubMed
    Score: 0.115
  2. Acetaminophen structure-toxicity studies: in vivo covalent binding of a nonhepatotoxic analog, 3-hydroxyacetanilide. Toxicol Appl Pharmacol. 1990 Sep 01; 105(2):195-208.
    View in: PubMed
    Score: 0.084
  3. Effects of sulfur-amino acid-deficient diets on acetaminophen metabolism and hepatotoxicity in rats. Toxicol Appl Pharmacol. 1989 Nov; 101(2):356-69.
    View in: PubMed
    Score: 0.079
  4. Effect of glucose and gluconeogenic substrates on fasting-induced suppression of acetaminophen glucuronidation in the rat. Biochem Pharmacol. 1989 Jan 15; 38(2):289-97.
    View in: PubMed
    Score: 0.075
  5. Mechanism of decreased acetaminophen glucuronidation in the fasted rat. Biochem Pharmacol. 1988 Mar 15; 37(6):1067-75.
    View in: PubMed
    Score: 0.070
  6. Relationship between sulfotransferase activity and susceptibility to acetaminophen-induced liver necrosis in the hamster. Drug Metab Dispos. 1987 Mar-Apr; 15(2):143-50.
    View in: PubMed
    Score: 0.066
  7. Mechanisms of fasting-induced potentiation of acetaminophen hepatotoxicity in the rat. Biochem Pharmacol. 1987 Feb 15; 36(4):427-33.
    View in: PubMed
    Score: 0.065
  8. Acetaminophen hepatotoxicity: studies on the mechanism of cysteamine protection. Toxicol Appl Pharmacol. 1986 Mar 30; 83(1):115-25.
    View in: PubMed
    Score: 0.062
  9. Anomalous susceptibility of the fasted hamster to acetaminophen hepatotoxicity. Biochem Pharmacol. 1986 Mar 01; 35(5):817-25.
    View in: PubMed
    Score: 0.061
  10. Strain differences in susceptibility of normal and diabetic rats to acetaminophen hepatotoxicity. Biochem Pharmacol. 1986 Feb 15; 35(4):687-95.
    View in: PubMed
    Score: 0.061
  11. The mechanisms of cobalt chloride-induced protection against acetaminophen hepatotoxicity. Drug Metab Dispos. 1986 Jan-Feb; 14(1):25-33.
    View in: PubMed
    Score: 0.061
  12. Mechanism of fasting-induced suppression of acetaminophen glucuronidation in the rat. Adv Exp Med Biol. 1986; 197:697-706.
    View in: PubMed
    Score: 0.061
  13. Role of UDPGA flux in acetaminophen clearance and hepatotoxicity. Xenobiotica. 1984 Jul; 14(7):553-9.
    View in: PubMed
    Score: 0.055
  14. Effect of L-ascorbic acid on acetaminophen-induced hepatotoxicity and covalent binding in hamsters. Evidence that in vitro covalent binding differs from that in vivo. Drug Metab Dispos. 1984 May-Jun; 12(3):271-9.
    View in: PubMed
    Score: 0.054
  15. Mechanism of ketone-induced protection from acetaminophen hepatotoxicity in the rat. Drug Metab Dispos. 1983 Sep-Oct; 11(5):451-7.
    View in: PubMed
    Score: 0.051
  16. Increased resistance of diabetic rats to acetaminophen-induced hepatotoxicity. J Pharmacol Exp Ther. 1982 Mar; 220(3):504-13.
    View in: PubMed
    Score: 0.046
  17. Biochemical basis for dose response relationships in reactive metabolite toxicity. Adv Exp Med Biol. 1981; 136 Pt A:99-113.
    View in: PubMed
    Score: 0.043
  18. Acetaminophen overdosage. J S C Med Assoc. 1977 Nov; 73(11):474-80.
    View in: PubMed
    Score: 0.034
  19. Acetaminophen-induced hepatic necrosis. VI. Metabolic disposition of toxic and nontoxic doses of acetaminophen. Pharmacology. 1974; 12(4-5):251-71.
    View in: PubMed
    Score: 0.026
  20. Acetaminophen-induced hepatic necrosis. II. Role of covalent binding in vivo. J Pharmacol Exp Ther. 1973 Oct; 187(1):195-202.
    View in: PubMed
    Score: 0.026
  21. Effect of cobalt protoporphyrin on hepatic drug-metabolizing enzymes. Specificity for cytochrome P-450. Biochem Pharmacol. 1989 Jun 15; 38(12):2027-38.
    View in: PubMed
    Score: 0.019
  22. Pharmacokinetic considerations in toxicity testing. Drug Metab Rev. 1982; 13(6):983-1007.
    View in: PubMed
    Score: 0.011
  23. Formation of chemically reactive metabolites of phenacetin and acetaminophen. Adv Exp Med Biol. 1981; 136 Pt B:931-50.
    View in: PubMed
    Score: 0.011
  24. Glutathione thresholds in reactive metabolite toxicity. Arch Toxicol Suppl. 1980; 3:95-110.
    View in: PubMed
    Score: 0.010
  25. Biochemical changes after hepatic injury from toxic doses of acetaminophen or furosemide. Pharmacology. 1976; 14(3):205-17.
    View in: PubMed
    Score: 0.008
  26. Acetaminophen-induced hepatic injury: protective role of glutathione in man and rationale for therapy. Clin Pharmacol Ther. 1974 Oct; 16(4):676-84.
    View in: PubMed
    Score: 0.007
  27. Species differences in hepatic glutathione depletion, covalent binding and hepatic necrosis after acetaminophen. Life Sci. 1974 Jun 01; 14(11):2099-109.
    View in: PubMed
    Score: 0.007
  28. Acetaminophen-induced hepatic necrosis. V. Correlation of hepatic necrosis, covalent binding and glutathione depletion in hamsters. Pharmacology. 1974; 12(3):129-43.
    View in: PubMed
    Score: 0.007
  29. Acetaminophen-induced hepatic necrosis. 3. Cytochrome P-450-mediated covalent binding in vitro. J Pharmacol Exp Ther. 1973 Oct; 187(1):203-10.
    View in: PubMed
    Score: 0.006
  30. Acetaminophen-induced hepatic necrosis. I. Role of drug metabolism. J Pharmacol Exp Ther. 1973 Oct; 187(1):185-94.
    View in: PubMed
    Score: 0.006
  31. Acetaminophen-induced hepatic necrosis. IV. Protective role of glutathione. J Pharmacol Exp Ther. 1973 Oct; 187(1):211-7.
    View in: PubMed
    Score: 0.006
  32. Diminished serum Gc (vitamin D-binding protein) levels and increased Gc:G-actin complexes in a hamster model of fulminant hepatic necrosis. Hepatology. 1987 Sep-Oct; 7(5):825-30.
    View in: PubMed
    Score: 0.004
  33. Drug metabolism as a cause of drug toxicity. Drug Metab Dispos. 1973 Jan-Feb; 1(1):418-23.
    View in: PubMed
    Score: 0.002
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.