Connection

David Jollow to Cricetinae

This is a "connection" page, showing publications David Jollow has written about Cricetinae.
Connection Strength

0.211
  1. Acetaminophen structure-toxicity studies: in vivo covalent binding of a nonhepatotoxic analog, 3-hydroxyacetanilide. Toxicol Appl Pharmacol. 1990 Sep 01; 105(2):195-208.
    View in: PubMed
    Score: 0.023
  2. Effect of cobalt protoporphyrin on hepatic drug-metabolizing enzymes. Specificity for cytochrome P-450. Biochem Pharmacol. 1989 Jun 15; 38(12):2027-38.
    View in: PubMed
    Score: 0.021
  3. Relationship between sulfotransferase activity and susceptibility to acetaminophen-induced liver necrosis in the hamster. Drug Metab Dispos. 1987 Mar-Apr; 15(2):143-50.
    View in: PubMed
    Score: 0.018
  4. Acetaminophen hepatotoxicity: studies on the mechanism of cysteamine protection. Toxicol Appl Pharmacol. 1986 Mar 30; 83(1):115-25.
    View in: PubMed
    Score: 0.017
  5. Anomalous susceptibility of the fasted hamster to acetaminophen hepatotoxicity. Biochem Pharmacol. 1986 Mar 01; 35(5):817-25.
    View in: PubMed
    Score: 0.017
  6. The mechanisms of cobalt chloride-induced protection against acetaminophen hepatotoxicity. Drug Metab Dispos. 1986 Jan-Feb; 14(1):25-33.
    View in: PubMed
    Score: 0.017
  7. Effect of L-ascorbic acid on acetaminophen-induced hepatotoxicity and covalent binding in hamsters. Evidence that in vitro covalent binding differs from that in vivo. Drug Metab Dispos. 1984 May-Jun; 12(3):271-9.
    View in: PubMed
    Score: 0.015
  8. Pharmacokinetic considerations in toxicity testing. Drug Metab Rev. 1982; 13(6):983-1007.
    View in: PubMed
    Score: 0.013
  9. Biochemical basis for dose response relationships in reactive metabolite toxicity. Adv Exp Med Biol. 1981; 136 Pt A:99-113.
    View in: PubMed
    Score: 0.012
  10. Glutathione thresholds in reactive metabolite toxicity. Arch Toxicol Suppl. 1980; 3:95-110.
    View in: PubMed
    Score: 0.011
  11. Metabolic activation of furosemide to a chemically reactive, hepatotoxic metabolite. J Pharmacol Exp Ther. 1976 Oct; 199(1):41-52.
    View in: PubMed
    Score: 0.009
  12. N-hydroxylation of p-chloroacetanilde in hamsters. Biochem Pharmacol. 1976 Mar 01; 25(5):599-601.
    View in: PubMed
    Score: 0.009
  13. Acetaminophen-induced hepatic necrosis. VI. Metabolic disposition of toxic and nontoxic doses of acetaminophen. Pharmacology. 1974; 12(4-5):251-71.
    View in: PubMed
    Score: 0.007
  14. Diminished serum Gc (vitamin D-binding protein) levels and increased Gc:G-actin complexes in a hamster model of fulminant hepatic necrosis. Hepatology. 1987 Sep-Oct; 7(5):825-30.
    View in: PubMed
    Score: 0.005
  15. Correlation between extent of liver damage in fulminant hepatic necrosis and complexing of circulating group-specific component (vitamin D-binding protein). J Lab Clin Med. 1987 Jul; 110(1):83-90.
    View in: PubMed
    Score: 0.005
  16. Vasopressin stimulates thromboxane synthesis in isolated hamster hepatocytes: relation to hepatocyte calcium content. Prostaglandins. 1983 Sep; 26(3):397-408.
    View in: PubMed
    Score: 0.004
  17. Formation of chemically reactive metabolites of phenacetin and acetaminophen. Adv Exp Med Biol. 1981; 136 Pt B:931-50.
    View in: PubMed
    Score: 0.003
  18. Species differences in hepatic glutathione depletion, covalent binding and hepatic necrosis after acetaminophen. Life Sci. 1974 Jun 01; 14(11):2099-109.
    View in: PubMed
    Score: 0.002
  19. Acetaminophen-induced hepatic necrosis. V. Correlation of hepatic necrosis, covalent binding and glutathione depletion in hamsters. Pharmacology. 1974; 12(3):129-43.
    View in: PubMed
    Score: 0.002
  20. The role of cytochrome P-450 in N-hydroxylation of 2-acetylaminofluorene. Mol Pharmacol. 1973 May; 9(3):398-404.
    View in: PubMed
    Score: 0.002
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.