Connection

Mitzi Nagarkatti to Mice, Knockout

This is a "connection" page, showing publications Mitzi Nagarkatti has written about Mice, Knockout.
Connection Strength

1.437
  1. CD44 deletion leading to attenuation of experimental autoimmune encephalomyelitis results from alterations in gut microbiome in mice. Eur J Immunol. 2017 07; 47(7):1188-1199.
    View in: PubMed
    Score: 0.106
  2. Dietary Indoles Suppress Delayed-Type Hypersensitivity by Inducing a Switch from Proinflammatory Th17 Cells to Anti-Inflammatory Regulatory T Cells through Regulation of MicroRNA. J Immunol. 2016 Feb 01; 196(3):1108-22.
    View in: PubMed
    Score: 0.096
  3. Critical Role of Mast Cells and Peroxisome Proliferator-Activated Receptor ? in the Induction of Myeloid-Derived Suppressor Cells by Marijuana Cannabidiol In Vivo. J Immunol. 2015 Jun 01; 194(11):5211-22.
    View in: PubMed
    Score: 0.091
  4. Staphylococcal enterotoxin B-induced microRNA-155 targets SOCS1 to promote acute inflammatory lung injury. Infect Immun. 2014 Jul; 82(7):2971-9.
    View in: PubMed
    Score: 0.085
  5. MicroRNA let-7e is associated with the pathogenesis of experimental autoimmune encephalomyelitis. Eur J Immunol. 2013 Jan; 43(1):104-14.
    View in: PubMed
    Score: 0.077
  6. Role of CD44 in the differentiation of Th1 and Th2 cells: CD44-deficiency enhances the development of Th2 effectors in response to sheep RBC and chicken ovalbumin. J Immunol. 2009 Jul 01; 183(1):172-80.
    View in: PubMed
    Score: 0.061
  7. Role of CD44 and its v7 isoform in staphylococcal enterotoxin B-induced toxic shock: CD44 deficiency on hepatic mononuclear cells leads to reduced activation-induced apoptosis that results in increased liver damage. Infect Immun. 2005 Jan; 73(1):50-61.
    View in: PubMed
    Score: 0.045
  8. Role of CD44 and hyaluronic acid (HA) in activation of alloreactive and antigen-specific T cells by bone marrow-derived dendritic cells. J Immunother. 2004 Jan-Feb; 27(1):1-12.
    View in: PubMed
    Score: 0.042
  9. Combined deficiency in CD44 and Fas leads to exacerbation of lymphoproliferative and autoimmune disease. Int Immunol. 2003 Nov; 15(11):1327-40.
    View in: PubMed
    Score: 0.041
  10. Targeted deletion of CD44v7 exon leads to decreased endothelial cell injury but not tumor cell killing mediated by interleukin-2-activated cytolytic lymphocytes. J Biol Chem. 2003 Oct 31; 278(44):43818-30.
    View in: PubMed
    Score: 0.040
  11. Environmental Microcystin exposure in underlying NAFLD-induced exacerbation of neuroinflammation, blood-brain barrier dysfunction, and neurodegeneration are NLRP3 and S100B dependent. Toxicology. 2021 09; 461:152901.
    View in: PubMed
    Score: 0.035
  12. Cannabinoid Receptor Activation on Haematopoietic Cells and Enterocytes Protects against Colitis. J Crohns Colitis. 2021 Jun 22; 15(6):1032-1048.
    View in: PubMed
    Score: 0.035
  13. CD44-deficient mice exhibit enhanced hepatitis after concanavalin A injection: evidence for involvement of CD44 in activation-induced cell death. J Immunol. 2001 May 15; 166(10):5889-97.
    View in: PubMed
    Score: 0.035
  14. AhR Ligands Differentially Regulate miRNA-132 Which Targets HMGB1 and to Control the Differentiation of Tregs and Th-17 Cells During Delayed-Type Hypersensitivity Response. Front Immunol. 2021; 12:635903.
    View in: PubMed
    Score: 0.034
  15. Autophagy Inhibition Enables Nrf2 to Exaggerate the Progression of Diabetic Cardiomyopathy in Mice. Diabetes. 2020 12; 69(12):2720-2734.
    View in: PubMed
    Score: 0.033
  16. Higher intestinal and circulatory lactate associated NOX2 activation leads to an ectopic fibrotic pathology following microcystin co-exposure in murine fatty liver disease. Comp Biochem Physiol C Toxicol Pharmacol. 2020 Dec; 238:108854.
    View in: PubMed
    Score: 0.033
  17. Molecules from American Ginseng Suppress Colitis through Nuclear Factor Erythroid-2-Related Factor 2. Nutrients. 2020 Jun 21; 12(6).
    View in: PubMed
    Score: 0.033
  18. Combination of Cannabinoids, ?9- Tetrahydrocannabinol and Cannabidiol, Ameliorates Experimental Multiple Sclerosis by Suppressing Neuroinflammation Through Regulation of miRNA-Mediated Signaling Pathways. Front Immunol. 2019; 10:1921.
    View in: PubMed
    Score: 0.031
  19. Exogenous PP2A inhibitor exacerbates the progression of nonalcoholic fatty liver disease via NOX2-dependent activation of miR21. Am J Physiol Gastrointest Liver Physiol. 2019 10 01; 317(4):G408-G428.
    View in: PubMed
    Score: 0.031
  20. Environmental microcystin targets the microbiome and increases the risk of intestinal inflammatory pathology via NOX2 in underlying murine model of Nonalcoholic Fatty Liver Disease. Sci Rep. 2019 06 19; 9(1):8742.
    View in: PubMed
    Score: 0.030
  21. Increased butyrate priming in the gut stalls microbiome associated-gastrointestinal inflammation and hepatic metabolic reprogramming in a mouse model of Gulf War Illness. Toxicol Appl Pharmacol. 2018 07 01; 350:64-77.
    View in: PubMed
    Score: 0.028
  22. High circulatory leptin mediated NOX-2-peroxynitrite-miR21 axis activate mesangial cells and promotes renal inflammatory pathology in nonalcoholic fatty liver disease. Redox Biol. 2018 07; 17:1-15.
    View in: PubMed
    Score: 0.028
  23. MDSCs drive the process of endometriosis by enhancing angiogenesis and are a new potential therapeutic target. Eur J Immunol. 2018 06; 48(6):1059-1073.
    View in: PubMed
    Score: 0.028
  24. Antibody blockade of CLEC12A delays EAE onset and attenuates disease severity by impairing myeloid cell CNS infiltration and restoring positive immunity. Sci Rep. 2017 06 02; 7(1):2707.
    View in: PubMed
    Score: 0.026
  25. Altered gut microbiome in a mouse model of Gulf War Illness causes neuroinflammation and intestinal injury via leaky gut and TLR4 activation. PLoS One. 2017; 12(3):e0172914.
    View in: PubMed
    Score: 0.026
  26. Nrf2-Mediated Cardiac Maladaptive Remodeling and Dysfunction in a Setting of Autophagy Insufficiency. Hypertension. 2016 Jan; 67(1):107-17.
    View in: PubMed
    Score: 0.024
  27. Identifying panaxynol, a natural activator of nuclear factor erythroid-2 related factor 2 (Nrf2) from American ginseng as a suppressor of inflamed macrophage-induced cardiomyocyte hypertrophy. J Ethnopharmacol. 2015 Jun 20; 168:326-36.
    View in: PubMed
    Score: 0.023
  28. Kruppel-like factor KLF4 facilitates cutaneous wound healing by promoting fibrocyte generation from myeloid-derived suppressor cells. J Invest Dermatol. 2015 May; 135(5):1425-1434.
    View in: PubMed
    Score: 0.022
  29. Micro-RNA 21 inhibition of SMAD7 enhances fibrogenesis via leptin-mediated NADPH oxidase in experimental and human nonalcoholic steatohepatitis. Am J Physiol Gastrointest Liver Physiol. 2015 Feb 15; 308(4):G298-312.
    View in: PubMed
    Score: 0.022
  30. miR-155 deficiency protects mice from experimental colitis by reducing T helper type 1/type 17 responses. Immunology. 2014 Nov; 143(3):478-89.
    View in: PubMed
    Score: 0.022
  31. Leptin antagonist ameliorates chronic colitis in IL-10?/? mice. Immunobiology. 2013 Dec; 218(12):1439-51.
    View in: PubMed
    Score: 0.020
  32. Cannabinoid receptor-2 (CB2) agonist ameliorates colitis in IL-10(-/-) mice by attenuating the activation of T cells and promoting their apoptosis. Toxicol Appl Pharmacol. 2012 Jan 15; 258(2):256-67.
    View in: PubMed
    Score: 0.018
  33. Activation of aryl hydrocarbon receptor (AhR) leads to reciprocal epigenetic regulation of FoxP3 and IL-17 expression and amelioration of experimental colitis. PLoS One. 2011; 6(8):e23522.
    View in: PubMed
    Score: 0.018
  34. Role of resveratrol-induced CD11b(+) Gr-1(+) myeloid derived suppressor cells (MDSCs) in the reduction of CXCR3(+) T cells and amelioration of chronic colitis in IL-10(-/-) mice. Brain Behav Immun. 2012 Jan; 26(1):72-82.
    View in: PubMed
    Score: 0.018
  35. Attenuation of experimental autoimmune hepatitis by exogenous and endogenous cannabinoids: involvement of regulatory T cells. Mol Pharmacol. 2008 Jul; 74(1):20-33.
    View in: PubMed
    Score: 0.014
  36. Primary peripheral T cells become susceptible to 2,3,7,8-tetrachlorodibenzo-p-dioxin-mediated apoptosis in vitro upon activation and in the presence of dendritic cells. Mol Pharmacol. 2008 Jun; 73(6):1722-35.
    View in: PubMed
    Score: 0.014
  37. Resveratrol (trans-3,5,4'-trihydroxystilbene) ameliorates experimental allergic encephalomyelitis, primarily via induction of apoptosis in T cells involving activation of aryl hydrocarbon receptor and estrogen receptor. Mol Pharmacol. 2007 Dec; 72(6):1508-21.
    View in: PubMed
    Score: 0.013
  38. Superantigen-primed T cells exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) replicate poorly following recall encounter. Arch Toxicol. 2006 Mar; 80(3):134-45.
    View in: PubMed
    Score: 0.012
  39. Treatment of mice with 2,3,7,8-tetrachlorodibenzo-p-dioxin leads to aryl hydrocarbon receptor-dependent nuclear translocation of NF-kappaB and expression of Fas ligand in thymic stromal cells and consequent apoptosis in T cells. J Immunol. 2005 Jul 01; 175(1):90-103.
    View in: PubMed
    Score: 0.012
  40. Aryl hydrocarbon receptor-dependent induction of loss of mitochondrial membrane potential in epididydimal spermatozoa by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Toxicol Lett. 2005 Jun 17; 157(2):99-107.
    View in: PubMed
    Score: 0.012
  41. Activation through cannabinoid receptors 1 and 2 on dendritic cells triggers NF-kappaB-dependent apoptosis: novel role for endogenous and exogenous cannabinoids in immunoregulation. J Immunol. 2004 Aug 15; 173(4):2373-82.
    View in: PubMed
    Score: 0.011
  42. Evidence for the involvement of CD44 in endothelial cell injury and induction of vascular leak syndrome by IL-2. J Immunol. 1999 Aug 01; 163(3):1619-27.
    View in: PubMed
    Score: 0.008
  43. Evidence for the involvement of Fas ligand and perforin in the induction of vascular leak syndrome. J Immunol. 1998 Sep 15; 161(6):3077-86.
    View in: PubMed
    Score: 0.007
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.