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Bell, P.
One or more keywords matched the following items that are connected to Bell, P.
Item TypeName
Academic Article Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors.
Academic Article Cilia movement regulates expression of the Raf-1 kinase inhibitor protein.
Academic Article Loss of primary cilia upregulates renal hypertrophic signaling and promotes cystogenesis.
Academic Article An inducible CiliaGFP mouse model for in vivo visualization and analysis of cilia in live tissue.
Academic Article TRPP2 and TRPV4 form an EGF-activated calcium permeable channel at the apical membrane of renal collecting duct cells.
Concept Cilia
Academic Article Deletion of airway cilia results in noninflammatory bronchiectasis and hyperreactive airways.
Academic Article The exocyst protein Sec10 interacts with Polycystin-2 and knockdown causes PKD-phenotypes.
Academic Article Epidermal growth factor-induced proliferation of collecting duct cells from Oak Ridge polycystic kidney mice involves activation of Na+/H+ exchanger.
Academic Article Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damage.
Academic Article Molecular pathways and therapies in autosomal-dominant polycystic kidney disease.
Academic Article Heightened epithelial Na+ channel-mediated Na+ absorption in a murine polycystic kidney disease model epithelium lacking apical monocilia.
Academic Article Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells.
Academic Article Loss of primary cilia increases polycystin-2 and TRPV4 and the appearance of a nonselective cation channel in the mouse cortical collecting duct.
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  • Cilia