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Pimelic diphenylamide 106 is a slow, tight-binding inhibitor of class I histone deacetylases.
Chemical probes identify a role for histone deacetylase 3 in Friedreich's ataxia gene silencing.
Two new pimelic diphenylamide HDAC inhibitors induce sustained frataxin upregulation in cells from Friedreich's ataxia patients and in a mouse model.
Mechanism of Action of 2-Aminobenzamide HDAC Inhibitors in Reversing Gene Silencing in Friedreich's Ataxia.