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Down-regulation of sphingosine kinase-1 by DNA damage: dependence on proteases and p53.
De novo ceramide regulates the alternative splicing of caspase 9 and Bcl-x in A549 lung adenocarcinoma cells. Dependence on protein phosphatase-1.
A mitochondrial pool of sphingomyelin is involved in TNFalpha-induced Bax translocation to mitochondria.
Anti-ceramidase LCL385 acutely reduces BCL-2 expression in the hippocampus but is not associated with an increase of learned helplessness in rats.
Proto-Oncogene Proteins c-bcl-2
Bcl-2 interrupts the ceramide-mediated pathway of cell death.
prICE: a downstream target for ceramide-induced apoptosis and for the inhibitory action of Bcl-2.
Cytokine response modifier A (CrmA) inhibits ceramide formation in response to tumor necrosis factor (TNF)-alpha: CrmA and Bcl-2 target distinct components in the apoptotic pathway.
Upregulation of Bcl-2 and elevation of ceramide in Batten disease.
Mechanisms of ceramide-mediated apoptosis.
Regulation of membrane release in apoptosis.
Inhibition of caspases inhibits the release of apoptotic bodies: Bcl-2 inhibits the initiation of formation of apoptotic bodies in chemotherapeutic agent-induced apoptosis.
Proteolytic cleavage of phospholipase C-gamma1 during apoptosis in Molt-4 cells.
Selective hydrolysis of a mitochondrial pool of sphingomyelin induces apoptosis.