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Loss of sphingosine kinase-1 activates the intrinsic pathway of programmed cell death: modulation of sphingolipid levels and the induction of apoptosis.
Ceramide generated by sphingomyelin hydrolysis and the salvage pathway is involved in hypoxia/reoxygenation-induced Bax redistribution to mitochondria in NT-2 cells.
Mitochondrially targeted ceramides preferentially promote autophagy, retard cell growth, and induce apoptosis.
A mitochondrial pool of sphingomyelin is involved in TNFalpha-induced Bax translocation to mitochondria.
AMPK inhibitor Compound C stimulates ceramide production and promotes Bax redistribution and apoptosis in MCF7 breast carcinoma cells.
bcl-2-Associated X Protein
bcl 2 Associated X Protein