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Effect of ras-gene transformation on the inhibition of NIH3T3 cell growth by pertussis toxin.
Differential regulation of G protein subunit expression in mouse oocytes, eggs, and early embryos.
ADP-ribosylation of membrane components by pertussis and cholera toxin.
Pertussis toxin alters the growth characteristics of Swiss 3T3 cells.
Pertussis toxin substrate, the putative Ni component of adenylyl cyclases, is an alpha beta heterodimer regulated by guanine nucleotide and magnesium.
Gi is involved in ethanol inhibition of L-type calcium channels in undifferentiated but not differentiated PC-12 cells.
Factors determining specificity of signal transduction by G-protein-coupled receptors. Regulation of signal transfer from receptor to G-protein.
Glucagon-induced heterologous desensitization of the MDCK cell adenylyl cyclase. Increases in the apparent levels of the inhibitory regulator (Ni).
The effect of pertussis toxin on the growth of vascular smooth muscle cells stimulated by serum or platelet-derived growth factor.
Virulence Factors, Bordetella
Virulence Factors Bordetella