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The mitochondrial permeability transition is required for tumor necrosis factor alpha-mediated apoptosis and cytochrome c release.
The mitochondrial permeability transition augments Fas-induced apoptosis in mouse hepatocytes.
NF-kappaB stimulates inducible nitric oxide synthase to protect mouse hepatocytes from TNF-alpha- and Fas-mediated apoptosis.
Mitochondrial permeability transition in the switch from necrotic to apoptotic cell death in ischemic rat hepatocytes.
Salicylate enhances necrosis and apoptosis mediated by the mitochondrial permeability transition.
Mitochondrial permeability transition in acetaminophen-induced necrosis and apoptosis of cultured mouse hepatocytes.
Graft tumor necrosis factor receptor-1 protects after mouse liver transplantation whereas host tumor necrosis factor receptor-1 promotes injury.
C-Jun N-terminal kinase 2 promotes graft injury via the mitochondrial permeability transition after mouse liver transplantation.
Differential effects of rapamycin on rods and cones during light-induced stress in albino mice.
Gliotoxin-mediated apoptosis of activated human hepatic stellate cells.
c-Jun N-terminal kinase mediates hepatic injury after rat liver transplantation.
Transforming growth factor beta mediates hepatocyte apoptosis through Smad3 generation of reactive oxygen species.
NIM811 prevents mitochondrial dysfunction, attenuates liver injury, and stimulates liver regeneration after massive hepatectomy.
Role of inducible nitric oxide synthase in mitochondrial depolarization and graft injury after transplantation of fatty livers.
Minocycline and doxycycline, but not tetracycline, mitigate liver and kidney injury after hemorrhagic shock/resuscitation.