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Thiamine deficiency in the pathogenesis of chronic ethanol-associated cerebellar damage in vitro.
Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures.
Ethanol disrupts NMDA receptor and astroglial EAAT2 modulation of Kv2.1 potassium channels in hippocampus.
Small conductance calcium-activated potassium type 2 channels regulate alcohol-associated plasticity of glutamatergic synapses.
NR2B-deficient mice are more sensitive to the locomotor stimulant and depressant effects of ethanol.
Glucocorticoid and polyamine interactions in the plasticity of glutamatergic synapses that contribute to ethanol-associated dependence and neuronal injury.
Central Nervous System Stimulants
Alcohol-Induced Disorders, Nervous System
Central Nervous System Depressants
KCNN Genes that Encode Small-Conductance Ca2+-Activated K+ Channels Influence Alcohol and Drug Addiction.
Chronic intermittent ethanol exposure and withdrawal leads to adaptations in nucleus accumbens core postsynaptic density proteome and dendritic spines.
Chronic intermittent ethanol induced axon and myelin degeneration is attenuated by calpain inhibition.