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Unique activation of extracellular striato-pallidal neurotransmitters in rats following acute risperidone.
Chronic haloperidol-induced alterations in pallidal GABA and striatal D(1)-mediated dopamine turnover as measured by dual probe microdialysis in rats.
Decreased pallidal GABA following reverse microdialysis with clozapine, but not haloperidol.
Convergent evidence from microdialysis and presynaptic immunolabeling for the regulation of gamma-aminobutyric acid release in the globus pallidus following acute clozapine or haloperidol administration in rats.
Chronic haloperidol, but not clozapine, produces altered oral movements and increased extracellular glutamate in rats.
Chronic neuroleptic administration decreases extracellular GABA in the nucleus accumbens but not in the caudate-putamen of rats.
Duration-dependent increase in striatal glutamate following prolonged fluphenazine administration in rats.
Chronic haloperidol potentiates stimulated glutamate release in caudate putamen, but not prefrontal cortex.
The neurotensin receptor antagonist SR 48692 decreases extracellular striatal GABA in rats.