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Brain Injury, Chronic
Identifying the Role of Complement in Triggering Neuroinflammation after Traumatic Brain Injury.
Acid sphingomyelinase deficiency protects mitochondria and improves function recovery after brain injury.
Anti-inflammatory and Neuroprotective Agents in Clinical Trials for CNS Disease and Injury: Where Do We Go From Here?
Complement Drives Synaptic Degeneration and Progressive Cognitive Decline in the Chronic Phase after Traumatic Brain Injury.
Complement mediates neuroinflammation and cognitive decline at extended chronic time points after traumatic brain injury.
Brain Injuries, Traumatic
Chronic complement dysregulation drives neuroinflammation after traumatic brain injury: a transcriptomic study.