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A complement-dependent balance between hepatic ischemia/reperfusion injury and liver regeneration in mice.
Oval cell response is attenuated by depletion of liver resident macrophages in the 2-AAF/partial hepatectomy rat.
Dissecting the complement pathway in hepatic injury and regeneration with a novel protective strategy.
Modulation of post-stroke degenerative and regenerative processes and subacute protection by site-targeted inhibition of the alternative pathway of complement.
Natural immunoglobulin M initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration in mice.
Inhibition of the alternative complement pathway accelerates repair processes in the murine model of choroidal neovascularization.