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Lipopolysaccharide sensitizes neonatal hypoxic-ischemic brain injury in a MyD88-dependent manner.
Inflammatory-induced hibernation in the fetus: priming of fetal sheep metabolism correlates with developmental brain injury.
The instrumented fetal sheep as a model of cerebral white matter injury in the premature infant.
Advanced magnetic resonance spectroscopy and imaging techniques applied to brain development and animal models of perinatal injury.
Complex interactions between hypoxia-ischemia and inflammation in preterm brain injury.
Perinatal brain injury: mechanisms and therapeutic approaches.
Human Umbilical Cord Blood Therapy Protects Cerebral White Matter from Systemic LPS Exposure in Preterm Fetal Sheep.
Anti-Inflammatory Therapies for Treatment of Inflammation-Related Preterm Brain Injury.
Cooling and immunomodulation for treating hypoxic-ischemic brain injury.
Back to the beginning: can we stop brain injury before it starts?