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Ontogeny of AMPA and NMDA receptor gene expression in the developing sheep white matter and cerebral cortex.
Timing of appearance of late oligodendrocyte progenitors coincides with enhanced susceptibility of preterm rabbit cerebral white matter to hypoxia-ischemia.
An organotypic slice culture model of chronic white matter injury with maturation arrest of oligodendrocyte progenitors.
Histopathological correlates of magnetic resonance imaging-defined chronic perinatal white matter injury.
The instrumented fetal sheep as a model of cerebral white matter injury in the premature infant.
Effect of cerebral hypothermia and asphyxia on the subventricular zone and white matter tracts in preterm fetal sheep.
High-field diffusion tensor imaging characterization of cerebral white matter injury in lipopolysaccharide-exposed fetal sheep.
Role of recurrent hypoxia-ischemia in preterm white matter injury severity.
Human Umbilical Cord Blood Therapy Protects Cerebral White Matter from Systemic LPS Exposure in Preterm Fetal Sheep.
Protection of axonal integrity with 48 or 72?h of cerebral hypothermia in near-term fetal sheep.
Long-term coordinated microstructural disruptions of the developing neocortex and subcortical white matter after early postnatal systemic inflammation.
Magnetic Resonance Imaging Correlates of White Matter Gliosis and Injury in Preterm Fetal Sheep Exposed to Progressive Systemic Inflammation.
Tumor necrosis factor inhibition attenuates white matter gliosis after systemic inflammation in preterm fetal sheep.
Interleukin-1 blockade attenuates white matter inflammation and oligodendrocyte loss after progressive systemic lipopolysaccharide exposure in near-term fetal sheep.