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Cui, Taixing
One or more keywords matched the following items that are connected to Cui, Taixing
Item TypeName
Concept Myocytes, Cardiac
Concept Myocytes, Smooth Muscle
Academic Article Up-regulation of p27(kip1) contributes to Nrf2-mediated protection against angiotensin II-induced cardiac hypertrophy.
Academic Article Nicotine enhances angiotensin II-induced mitogenic response in vascular smooth muscle cells and fibroblasts.
Academic Article Regulation of inhibitory protein-kappaB and monocyte chemoattractant protein-1 by angiotensin II type 2 receptor-activated Src homology protein tyrosine phosphatase-1 in fetal vascular smooth muscle cells.
Academic Article Effect of azelnidipine on angiotensin II-mediated growth-promoting signaling in vascular smooth muscle cells.
Academic Article Rad GTPase attenuates vascular lesion formation by inhibition of vascular smooth muscle cell migration.
Academic Article Impaired expression of PPAR gamma protein contributes to the exaggerated growth of vascular smooth muscle cells in spontaneously hypertensive rats.
Academic Article Nitro-linoleic acid inhibits vascular smooth muscle cell proliferation via the Keap1/Nrf2 signaling pathway.
Academic Article Ubiquitin carboxyl-terminal hydrolase L1, a novel deubiquitinating enzyme in the vasculature, attenuates NF-kappaB activation.
Academic Article A highly efficient method to differentiate smooth muscle cells from human embryonic stem cells.
Academic Article Rad GTPase deficiency leads to cardiac hypertrophy.
Academic Article Nrf2 protects against maladaptive cardiac responses to hemodynamic stress.
Academic Article Ubiquitin carboxyl terminal hydrolase L1 negatively regulates TNFalpha-mediated vascular smooth muscle cell proliferation via suppressing ERK activation.
Academic Article Dihydro-CDDO-trifluoroethyl amide (dh404), a novel Nrf2 activator, suppresses oxidative stress in cardiomyocytes.
Academic Article An essential role of Nrf2 in American ginseng-mediated anti-oxidative actions in cardiomyocytes.
Academic Article Diabetic downregulation of Nrf2 activity via ERK contributes to oxidative stress-induced insulin resistance in cardiac cells in vitro and in vivo.
Academic Article A pro-inflammatory role of deubiquitinating enzyme cylindromatosis (CYLD) in vascular smooth muscle cells.
Academic Article American ginseng inhibits vascular smooth muscle cell proliferation via suppressing Jak/Stat pathway.
Academic Article Nrf2 enhances myocardial clearance of toxic ubiquitinated proteins.
Academic Article Identifying panaxynol, a natural activator of nuclear factor erythroid-2 related factor 2 (Nrf2) from American ginseng as a suppressor of inflamed macrophage-induced cardiomyocyte hypertrophy.
Academic Article Deubiquitinating enzyme CYLD mediates pressure overload-induced cardiac maladaptive remodeling and dysfunction via downregulating Nrf2.
Academic Article A critical role of cardiac fibroblast-derived exosomes in activating renin angiotensin system in cardiomyocytes.
Academic Article Nrf2-Mediated Cardiac Maladaptive Remodeling and Dysfunction in a Setting of Autophagy Insufficiency.
Academic Article Nuclear factor erythroid-2 related factor 2 (Nrf2)-mediated protein quality control in cardiomyocytes.
Academic Article TFEB activation protects against cardiac proteotoxicity via increasing autophagic flux.
Academic Article Inhibitory role of reactive oxygen species in the differentiation of multipotent vascular stem cells into vascular smooth muscle cells in rats: a novel aspect of traditional culture of rat aortic smooth muscle cells.
Academic Article The Calcineurin-TFEB-p62 Pathway Mediates the Activation of Cardiac Macroautophagy by Proteasomal Malfunction.
Academic Article Mature Vascular Smooth Muscle Cells, but Not Endothelial Cells, Serve as the Major Cellular Source of Intimal Hyperplasia in Vein Grafts.
Academic Article CYLD exaggerates pressure overload-induced cardiomyopathy via suppressing autolysosome efflux in cardiomyocytes.
Academic Article An inhibitor role of Nrf2 in the regulation of myocardial senescence and dysfunction after myocardial infarction.
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  • Muscle Cells