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Myocyte contractility with caspase inhibition and simulated hyperkalemic cardioplegic arrest.
Trafficking of the membrane type-1 matrix metalloproteinase in ischemia and reperfusion: relation to interstitial membrane type-1 matrix metalloproteinase activity.
Matrix metalloproteinase-7 affects connexin-43 levels, electrical conduction, and survival after myocardial infarction.
Protein kinase C isoform activation and endothelin-1 mediated defects in myocyte contractility after cardioplegic arrest and reperfusion.
Differential effects of protein kinase C isoform activation in endothelin-mediated myocyte contractile dysfunction with cardioplegic arrest and reperfusion.
Changes in extracellular collagen matrix alter myocardial systolic performance.
Effects of adrenomedullin on human myocyte contractile function and beta-adrenergic response.
Left ventricular form and function: scientific priorities and strategic planning for development of new views of disease.
Caspase inhibition attenuates contractile dysfunction following cardioplegic arrest and rewarming in the setting of left ventricular failure.
Cardiac support device modifies left ventricular geometry and myocardial structure after myocardial infarction.
Mitral regurgitation augments post-myocardial infarction remodeling failure of hypertrophic compensation.
Caspase inhibition modulates left ventricular remodeling following myocardial infarction through cellular and extracellular mechanisms.
Basigin Promotes Cardiac Fibrosis and Failure in Response to Chronic Pressure Overload in Mice.