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Grabowski, Gregory
One or more keywords matched the following items that are connected to Grabowski, Gregory
Item TypeName
Concept beta-Glucosidase
Academic Article Involvement of acid beta-glucosidase 1 in the salvage pathway of ceramide formation.
Academic Article Saposin C is required for normal resistance of acid beta-glucosidase to proteolytic degradation.
Academic Article Viable mouse models of acid beta-glucosidase deficiency: the defect in Gaucher disease.
Academic Article Translation modulation of acid beta-glucosidase in HepG2 cells: participation of the PKC pathway.
Academic Article Gaucher disease mouse models: point mutations at the acid beta-glucosidase locus combined with low-level prosaposin expression lead to disease variants.
Academic Article Conditional expression of human acid beta-glucosidase improves the visceral phenotype in a Gaucher disease mouse model.
Academic Article Dependence of reversibility and progression of mouse neuronopathic Gaucher disease on acid beta-glucosidase residual activity levels.
Academic Article Participation of asparagine 370 and glutamine 235 in the catalysis by acid beta-glucosidase: the enzyme deficient in Gaucher disease.
Academic Article Acid beta-glucosidase 1 counteracts p38delta-dependent induction of interleukin-6: possible role for ceramide as an anti-inflammatory lipid.
Academic Article In vivo and ex vivo evaluation of L-type calcium channel blockers on acid beta-glucosidase in Gaucher disease mouse models.
Academic Article Specific saposin C deficiency: CNS impairment and acid beta-glucosidase effects in the mouse.
Academic Article Acid ?-glucosidase mutants linked to Gaucher disease, Parkinson disease, and Lewy body dementia alter a-synuclein processing.
Academic Article Ex vivo and in vivo effects of isofagomine on acid ?-glucosidase variants and substrate levels in Gaucher disease.
Academic Article Multiple pathogenic proteins implicated in neuronopathic Gaucher disease mice.
Academic Article The LIMP-2/SCARB2 binding motif on acid ?-glucosidase: basic and applied implications for Gaucher disease and associated neurodegenerative diseases.
Academic Article Combination of acid ?-glucosidase mutation and Saposin C deficiency in mice reveals Gba1 mutation dependent and tissue-specific disease phenotype.
Academic Article Intravenous infusion of iPSC-derived neural precursor cells increases acid ?-glucosidase function in the brain and lessens the neuronopathic phenotype in a mouse model of Gaucher disease.
Academic Article Ex vivo localization of the mouse saposin C activation region for acid beta-glucosidase.
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  • Glucosidases