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Insulin-Like Growth Factor Binding Protein 5
Insulin-Like Growth Factor Binding Protein 2
Insulin-Like Growth Factor Binding Protein 3
Insulin-like growth factor binding proteins 3 and 5 are overexpressed in idiopathic pulmonary fibrosis and contribute to extracellular matrix deposition.
Insulin-like growth factor binding protein 5 induces skin fibrosis: A novel murine model for dermal fibrosis.
Insulin-like growth factor-binding protein-5 induces pulmonary fibrosis and triggers mononuclear cellular infiltration.
The pro-fibrotic factor IGFBP-5 induces lung fibroblast and mononuclear cell migration.
Role of insulin-like growth factor binding protein-3 in allergic airway remodeling.
The fibrotic phenotype induced by IGFBP-5 is regulated by MAPK activation and egr-1-dependent and -independent mechanisms.
Decreased caveolin-1 levels contribute to fibrosis and deposition of extracellular IGFBP-5.
Localized expression of tenascin in systemic sclerosis-associated pulmonary fibrosis and its regulation by insulin-like growth factor binding protein 3.
Syndecan-2 is a novel target of insulin-like growth factor binding protein-3 and is over-expressed in fibrosis.
The membrane-associated adaptor protein DOK5 is upregulated in systemic sclerosis and associated with IGFBP-5-induced fibrosis.
IGFBP-5 Promotes Fibrosis Independently of Its Translocation to the Nucleus and Its Interaction with Nucleolin and IGF.
IGFBP-5 Promotes Fibrosis via Increasing Its Own Expression and That of Other Pro-fibrotic Mediators.
Intracellular Heat Shock Protein 70 Deficiency in Pulmonary Fibrosis.
NADPH oxidase-mediated induction of reactive oxygen species and extracellular matrix deposition by insulin-like growth factor binding protein-5.
Insulin-like growth factor binding protein-4 exerts antifibrotic activity by reducing levels of connective tissue growth factor and the C-X-C chemokine receptor 4.
Phenotypic Characterization of Transgenic Mice Expressing Human IGFBP-5.
Prominence of IL6, IGF, TLR, and Bioenergetics Pathway Perturbation in Lung Tissues of Scleroderma Patients With Pulmonary Fibrosis.
Identification of Impacted Pathways and Transcriptomic Markers as Potential Mediators of Pulmonary Fibrosis in Transgenic Mice Expressing Human IGFBP5.
Insulin Like Growth Factor Binding Protein 2