Home
About
Overview
Sharing Data
ORCID
Help
History (1)
Akt promotes tumorigenesis in part through modulating genomic instability via phosphorylating XLF.
See All Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
Akt promotes tumorigenesis in part through modulating genomic instability via phosphorylating XLF.
Akt promotes tumorigenesis in part through modulating genomic instability via phosphorylating XLF. Nucleus. 2015; 6(4):261-5.
View in:
PubMed
subject areas
Amino Acid Sequence
Carcinogenesis
DNA Damage
DNA Ligase ATP
DNA Ligases
DNA Repair
DNA Repair Enzymes
DNA-Binding Proteins
Genomic Instability
Homologous Recombination
Humans
Molecular Sequence Data
Phosphorylation
Promoter Regions, Genetic
Proto-Oncogene Proteins c-akt
Stress, Physiological
authors with profiles
Wenjian Gan