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Akt promotes tumorigenesis in part through modulating genomic instability via phosphorylating XLF.
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Akt promotes tumorigenesis in part through modulating genomic instability via phosphorylating XLF.
Akt promotes tumorigenesis in part through modulating genomic instability via phosphorylating XLF. Nucleus. 2015; 6(4):261-5.
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PubMed
subject areas
Amino Acid Sequence
Carcinogenesis
DNA Damage
DNA Ligase ATP
DNA Ligases
DNA Repair
DNA Repair Enzymes
DNA-Binding Proteins
Genomic Instability
Homologous Recombination
Humans
Molecular Sequence Data
Phosphorylation
Promoter Regions, Genetic
Proto-Oncogene Proteins c-akt
Stress, Physiological
authors with profiles
Wenjian Gan