9,10-Dimethyl-1,2-benzanthracene
                             
                            
                            
                                
                            
                            
                                
                            
                            
                            
                                
                                    
                                            
	"9,10-Dimethyl-1,2-benzanthracene" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, 
	MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, 
	which enables searching at various levels of specificity.
	
	
		
			
			
				7,12-Dimethylbenzanthracene. Polycyclic aromatic hydrocarbon found in tobacco smoke that is a potent carcinogen.
    
			
			
				
				
					
						| Descriptor ID | D015127 | 
					
						| MeSH Number(s) | D02.455.426.559.847.149.301 D04.615.149.301 | 
					
						| Concept/Terms |  | 
					
				
			 
			
				Below are MeSH descriptors whose meaning is more general than "9,10-Dimethyl-1,2-benzanthracene".
				
			 
			
			
				Below are MeSH descriptors whose meaning is more specific than "9,10-Dimethyl-1,2-benzanthracene".
				
			 
		 
	 
 
                                        
                                            
	
	
		
			
			
					
				This graph shows the total number of publications written about "9,10-Dimethyl-1,2-benzanthracene" by people in this website by year, and whether "9,10-Dimethyl-1,2-benzanthracene" was a major or minor topic of these publications. 
				
					 
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		            | Year | Major Topic | Minor Topic | Total | 
|---|
| 1997 | 0 | 1 | 1 | 
| 2004 | 0 | 1 | 1 | 
| 2007 | 0 | 1 | 1 | 
| 2011 | 0 | 2 | 2 | 
| 2013 | 0 | 1 | 1 | 
| 2019 | 0 | 1 | 1 | 
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				Below are the most recent publications written about "9,10-Dimethyl-1,2-benzanthracene" by people in Profiles.
						
					
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								Different oral cancer scenarios to personalize targeted therapy: Boron Neutron Capture Therapy translational studies. Ther Deliv. 2019 06 01; 10(6):353-362. 
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								Elevated ornithine decarboxylase activity promotes skin tumorigenesis by stimulating the recruitment of bulge stem cells but not via toxic polyamine catabolic metabolites. Amino Acids. 2014 Mar; 46(3):543-52. 
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								Quantification of epithelial cell differentiation in mammary glands and carcinomas from DMBA- and MNU-exposed rats. PLoS One. 2011; 6(10):e26145. 
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								The non-protein coding breast cancer susceptibility locus Mcs5a acts in a non-mammary cell-autonomous fashion through the immune system and modulates T-cell homeostasis and functions. Breast Cancer Res. 2011 Aug 16; 13(4):R81. 
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								Studies of the mechanism by which increased spermidine/spermine N1-acetyltransferase activity increases susceptibility to skin carcinogenesis. Carcinogenesis. 2007 Nov; 28(11):2404-11. 
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								Differential regulation of a fibroblast growth factor-binding protein during skin carcinogenesis and wound healing. Neoplasia. 2004 Sep-Oct; 6(5):595-602. 
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								Carcinogenic polycyclic aromatic hydrocarbons increase intracellular Ca2+ and cell proliferation in primary human mammary epithelial cells. Carcinogenesis. 1997 Jun; 18(6):1177-82. 
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								Insulin like growth factor-I independence in rat mammary carcinoma cells: a dominant phenotype in somatic cell hybrid experiments. Cancer Lett. 1993 Nov 01; 74(3):189-95. 
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								The role of Ha-ras oncogenes in growth factor independence in rat mammary carcinoma cells. Mol Carcinog. 1991; 4(4):286-96. 
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								Importance of extended growth potential and growth factor independence on in vivo neoplastic potential of primary rat mammary carcinoma cells. Cancer Res. 1987 Oct 15; 47(20):5316-22.