Connection

Dennis Watson to Nuclear Proteins

This is a "connection" page, showing publications Dennis Watson has written about Nuclear Proteins.
Connection Strength

0.801
  1. SP100 inhibits ETS1 activity in primary endothelial cells. Oncogene. 2005 Jan 27; 24(5):916-31.
    View in: PubMed
    Score: 0.212
  2. SP100 expression modulates ETS1 transcriptional activity and inhibits cell invasion. Oncogene. 2004 Aug 26; 23(39):6654-65.
    View in: PubMed
    Score: 0.206
  3. FLI1 and EWS-FLI1 function as ternary complex factors and ELK1 and SAP1a function as ternary and quaternary complex factors on the Egr1 promoter serum response elements. Oncogene. 1997 Jan 16; 14(2):213-21.
    View in: PubMed
    Score: 0.122
  4. Abnormal expression of FLI1 protein is an adverse prognostic factor in acute myeloid leukemia. Blood. 2011 Nov 17; 118(20):5604-12.
    View in: PubMed
    Score: 0.084
  5. EAPII interacts with ETS1 and modulates its transcriptional function. Oncogene. 2003 May 08; 22(18):2699-709.
    View in: PubMed
    Score: 0.047
  6. Regulation of Ets function by protein - protein interactions. Oncogene. 2000 Dec 18; 19(55):6514-23.
    View in: PubMed
    Score: 0.040
  7. EAP1/Daxx interacts with ETS1 and represses transcriptional activation of ETS1 target genes. Oncogene. 2000 Feb 10; 19(6):745-53.
    View in: PubMed
    Score: 0.038
  8. Functional relationships among ETS gene family members. Leukemia. 1997 Apr; 11 Suppl 3:557-66.
    View in: PubMed
    Score: 0.031
  9. Alterations in PMS2, MSH2 and MLH1 expression in human prostate cancer. Int J Oncol. 2003 May; 22(5):1033-43.
    View in: PubMed
    Score: 0.012
  10. Defects of DNA mismatch repair in human prostate cancer. Cancer Res. 2001 May 15; 61(10):4112-21.
    View in: PubMed
    Score: 0.010
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.