Connection

John Woodward to Recombinant Proteins

This is a "connection" page, showing publications John Woodward has written about Recombinant Proteins.
Connection Strength

1.357
  1. Dephosphorylation of GluN2B C-terminal tyrosine residues does not contribute to acute ethanol inhibition of recombinant NMDA receptors. Alcohol. 2013 May; 47(3):181-6.
    View in: PubMed
    Score: 0.375
  2. Enhanced ethanol inhibition of recombinant N-methyl-D-aspartate receptors by magnesium: role of NR3A subunits. Alcohol Clin Exp Res. 2008 Jun; 32(6):1059-66.
    View in: PubMed
    Score: 0.269
  3. Effects of ethanol on phosphorylation site mutants of recombinant N-methyl-D-aspartate receptors. Alcohol. 2011 Jun; 45(4):373-80.
    View in: PubMed
    Score: 0.081
  4. Expression of glycine-activated diheteromeric NR1/NR3 receptors in human embryonic kidney 293 cells Is NR1 splice variant-dependent. J Pharmacol Exp Ther. 2009 Dec; 331(3):975-84.
    View in: PubMed
    Score: 0.074
  5. Ethanol inhibition of recombinant NMDA receptors is not altered by coexpression of CaMKII-alpha or CaMKII-beta. Alcohol. 2008 Aug; 42(5):425-32.
    View in: PubMed
    Score: 0.068
  6. Effects of 8 different NR1 splice variants on the ethanol inhibition of recombinant NMDA receptors. Alcohol Clin Exp Res. 2006 Apr; 30(4):673-9.
    View in: PubMed
    Score: 0.058
  7. Effects of amino acid substitutions in transmembrane domains of the NR1 subunit on the ethanol inhibition of recombinant N-methyl-D-aspartate receptors. Alcohol Clin Exp Res. 2006 Mar; 30(3):523-30.
    View in: PubMed
    Score: 0.058
  8. Effect of the NR3 subunit on ethanol inhibition of recombinant NMDA receptors. Brain Res. 2003 Oct 10; 987(1):117-21.
    View in: PubMed
    Score: 0.049
  9. Ethanol inhibition of recombinant NR1/2A receptors: effects of heavy metal chelators and a zinc-insensitive NR2A mutant. Alcohol. 2003 Aug-Oct; 31(1-2):71-6.
    View in: PubMed
    Score: 0.049
  10. Effects of volatile solvents on recombinant N-methyl-D-aspartate receptors expressed in Xenopus oocytes. Br J Pharmacol. 2000 Dec; 131(7):1303-8.
    View in: PubMed
    Score: 0.040
  11. Reduced ethanol inhibition of N-methyl-D-aspartate receptors by deletion of the NR1 C0 domain or overexpression of alpha-actinin-2 proteins. J Biol Chem. 2000 May 19; 275(20):15019-24.
    View in: PubMed
    Score: 0.039
  12. Fyn tyrosine kinase reduces the ethanol inhibition of recombinant NR1/NR2A but not NR1/NR2B NMDA receptors expressed in HEK 293 cells. J Neurochem. 1999 Apr; 72(4):1389-93.
    View in: PubMed
    Score: 0.036
  13. Effects of c-Src tyrosine kinase on ethanol sensitivity of recombinant NMDA receptors expressed in HEK 293 cells. Alcohol Clin Exp Res. 1999 Feb; 23(2):357-62.
    View in: PubMed
    Score: 0.036
  14. Intracellular calcium enhances the ethanol sensitivity of NMDA receptors through an interaction with the C0 domain of the NR1 subunit. J Neurochem. 1998 Sep; 71(3):1095-107.
    View in: PubMed
    Score: 0.035
  15. Effects of the abused solvent toluene on recombinant N-methyl-D-aspartate and non-N-methyl-D-aspartate receptors expressed in Xenopus oocytes. J Pharmacol Exp Ther. 1998 Jul; 286(1):334-40.
    View in: PubMed
    Score: 0.034
  16. Effects of acute and chronic ethanol exposure on heteromeric N-methyl-D-aspartate receptors expressed in HEK 293 cells. J Neurochem. 1997 Dec; 69(6):2345-54.
    View in: PubMed
    Score: 0.033
  17. Ethanol differentially affects ATP-gated P2X(3) and P2X(4) receptor subtypes expressed in Xenopus oocytes. Neuropharmacology. 2005 Aug; 49(2):243-53.
    View in: PubMed
    Score: 0.014
  18. Activation of mitogen-activated protein kinases is required for alpha1-adrenergic agonist-induced cell scattering in transfected HepG2 cells. Exp Cell Res. 2000 Jul 10; 258(1):109-20.
    View in: PubMed
    Score: 0.010
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.