NK Cell Lectin-Like Receptor Subfamily K
"NK Cell Lectin-Like Receptor Subfamily K" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An activating NK cell lectin-like receptor subfamily that regulates immune responses to INFECTION and NEOPLASMS. Members of this subfamily generally occur as homodimers.
| Descriptor ID |
D055655
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| MeSH Number(s) |
D12.776.543.750.705.895.800.910
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| Concept/Terms |
NK Cell Lectin-Like Receptor Subfamily K- NK Cell Lectin-Like Receptor Subfamily K
- NK Cell Lectin Like Receptor Subfamily K
- Killer Cell Lectin-Like Receptor Subfamily K
- Killer Cell Lectin Like Receptor Subfamily K
NK Cell Lectin-Like Receptor Subfamily K, Member 1- NK Cell Lectin-Like Receptor Subfamily K, Member 1
- NK Cell Lectin Like Receptor Subfamily K, Member 1
- NKG2D Receptor
- Receptor, NKG2D
- CD314 Antigen
- Antigen, CD314
- NKG2-D Receptor
- NKG2 D Receptor
- Receptor, NKG2-D
- Killer Cell Lectin-Like Receptor Subfamily K, Member 1
- Killer Cell Lectin Like Receptor Subfamily K, Member 1
- NK Cell Receptor D
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Below are MeSH descriptors whose meaning is more general than "NK Cell Lectin-Like Receptor Subfamily K".
Below are MeSH descriptors whose meaning is more specific than "NK Cell Lectin-Like Receptor Subfamily K".
This graph shows the total number of publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in this website by year, and whether "NK Cell Lectin-Like Receptor Subfamily K" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2013 | 1 | 0 | 1 |
| 2014 | 0 | 1 | 1 |
| 2017 | 0 | 2 | 2 |
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Below are the most recent publications written about "NK Cell Lectin-Like Receptor Subfamily K" by people in Profiles.
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Antibody-mediated neutralization of soluble MIC significantly enhances CTLA4 blockade therapy. Sci Adv. 2017 May; 3(5):e1602133.
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Increased Natural Killer Cell Activation in HIV-Infected Immunologic Non-Responders Correlates with CD4+ T Cell Recovery after Antiretroviral Therapy and Viral Suppression. PLoS One. 2017; 12(1):e0167640.
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T-bet is critical for the development of acute graft-versus-host disease through controlling T cell differentiation and function. J Immunol. 2015 Jan 01; 194(1):388-97.
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NKG2D blockade inhibits poly(I:C)-triggered fetal loss in wild type but not in IL-10-/- mice. J Immunol. 2013 Apr 01; 190(7):3639-47.