Glucose Transporter Type 4
"Glucose Transporter Type 4" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A glucose transport protein found in mature MUSCLE CELLS and ADIPOCYTES. It promotes transport of glucose from the BLOOD into target TISSUES. The inactive form of the protein is localized in CYTOPLASMIC VESICLES. In response to INSULIN, it is translocated to the PLASMA MEMBRANE where it facilitates glucose uptake.
| Descriptor ID |
D051275
|
| MeSH Number(s) |
D12.776.157.530.500.500.937 D12.776.157.530.937.563.937 D12.776.543.585.500.500.937 D12.776.543.585.937.625.937
|
| Concept/Terms |
Glucose Transporter Type 4- Glucose Transporter Type 4
- Insulin-Responsive Glucose Transporter
- Glucose Transporter, Insulin-Responsive
- Insulin Responsive Glucose Transporter
- SLC2A4 Protein
- Solute Carrier Family 2, Facilitated Glucose Transporter, Member 4 Protein
- GLUT4 Protein
- GLUT-4 Protein
- GLUT 4 Protein
|
Below are MeSH descriptors whose meaning is more general than "Glucose Transporter Type 4".
Below are MeSH descriptors whose meaning is more specific than "Glucose Transporter Type 4".
This graph shows the total number of publications written about "Glucose Transporter Type 4" by people in this website by year, and whether "Glucose Transporter Type 4" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 0 | 1 | 1 |
| 1995 | 0 | 3 | 3 |
| 1996 | 0 | 1 | 1 |
| 1997 | 0 | 1 | 1 |
| 1998 | 0 | 1 | 1 |
| 1999 | 0 | 1 | 1 |
| 2001 | 0 | 2 | 2 |
| 2002 | 0 | 2 | 2 |
| 2004 | 0 | 1 | 1 |
| 2005 | 0 | 1 | 1 |
| 2007 | 1 | 3 | 4 |
| 2009 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2013 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
| 2015 | 1 | 0 | 1 |
| 2017 | 1 | 0 | 1 |
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Below are the most recent publications written about "Glucose Transporter Type 4" by people in Profiles.
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Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stresses. Am J Physiol Heart Circ Physiol. 2017 Dec 01; 313(6):H1098-H1108.
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Purinergic receptor X7 mediates leptin induced GLUT4 function in stellate cells in nonalcoholic steatohepatitis. Biochim Biophys Acta. 2016 01; 1862(1):32-45.
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Impact of the controlled release of a connexin 43 peptide on corneal wound closure in an STZ model of type I diabetes. PLoS One. 2014; 9(1):e86570.
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6-Mercaptopurine augments glucose transport activity in skeletal muscle cells in part via a mechanism dependent upon orphan nuclear receptor NR4A3. Am J Physiol Endocrinol Metab. 2013 Nov 01; 305(9):E1081-92.
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Oncogene activation induces metabolic transformation resulting in insulin-independence in human breast cancer cells. PLoS One. 2011 Mar 17; 6(3):e17959.
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Impaired insulin signaling accelerates cardiac mitochondrial dysfunction after myocardial infarction. J Mol Cell Cardiol. 2009 Jun; 46(6):910-8.
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NR4A orphan nuclear receptors modulate insulin action and the glucose transport system: potential role in insulin resistance. J Biol Chem. 2007 Oct 26; 282(43):31525-33.
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Lentivirus-mediated downregulation of hypothalamic insulin receptor expression. Physiol Behav. 2007 Nov 23; 92(4):691-701.
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Corticosterone impairs insulin-stimulated translocation of GLUT4 in the rat hippocampus. Neuroendocrinology. 2007; 85(2):71-80.
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Transgenerational inheritance of the insulin-resistant phenotype in embryo-transferred intrauterine growth-restricted adult female rat offspring. Am J Physiol Endocrinol Metab. 2007 May; 292(5):E1270-9.