"GABA Agonists" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Endogenous compounds and drugs that bind to and activate GAMMA-AMINOBUTYRIC ACID receptors (RECEPTORS, GABA).
| Descriptor ID |
D018755
|
| MeSH Number(s) |
D27.505.519.625.240.200 D27.505.696.577.240.200
|
| Concept/Terms |
GABA Agonists- GABA Agonists
- Agonists, GABA
- gamma-Aminobutyric Acid Agonists
- Acid Agonists, gamma-Aminobutyric
- Agonists, gamma-Aminobutyric Acid
- gamma Aminobutyric Acid Agonists
- GABA Receptor Agonists
- Agonists, GABA Receptor
- Receptor Agonists, GABA
- gamma-Aminobutyric Acid Agonist
- Acid Agonist, gamma-Aminobutyric
- Agonist, gamma-Aminobutyric Acid
- gamma Aminobutyric Acid Agonist
- GABA Agonist
- Agonist, GABA
- GABA Receptor Agonist
- Agonist, GABA Receptor
- Receptor Agonist, GABA
|
Below are MeSH descriptors whose meaning is more general than "GABA Agonists".
Below are MeSH descriptors whose meaning is more specific than "GABA Agonists".
This graph shows the total number of publications written about "GABA Agonists" by people in this website by year, and whether "GABA Agonists" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2003 | 1 | 2 | 3 |
| 2004 | 0 | 2 | 2 |
| 2005 | 2 | 0 | 2 |
| 2006 | 1 | 1 | 2 |
| 2007 | 1 | 1 | 2 |
| 2008 | 0 | 1 | 1 |
| 2010 | 1 | 1 | 2 |
| 2013 | 0 | 2 | 2 |
| 2015 | 0 | 1 | 1 |
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Below are the most recent publications written about "GABA Agonists" by people in Profiles.
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Tonic Inhibitory Control of Dentate Gyrus Granule Cells by a5-Containing GABAA Receptors Reduces Memory Interference. J Neurosci. 2015 Oct 07; 35(40):13698-712.
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Inactivation of the lateral habenula reduces anxiogenic behavior and cocaine seeking under conditions of heightened stress. Pharmacol Biochem Behav. 2013 Oct; 111:24-9.
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Relapse induced by cues predicting cocaine depends on rapid, transient synaptic potentiation. Neuron. 2013 Mar 06; 77(5):867-72.
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The infralimbic cortex regulates the consolidation of extinction after cocaine self-administration. Learn Mem. 2010 Apr; 17(4):168-75.
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Activation of alpha6-containing GABAA receptors by pentobarbital occurs through a different mechanism than activation by GABA. Neurosci Lett. 2010 Mar 08; 471(3):195-9.
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The glutamatergic projection from the prefrontal cortex to the nucleus accumbens core is required for cocaine-induced decreases in ventral pallidal GABA. Neurosci Lett. 2008 Jun 20; 438(2):142-5.
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Microscopic kinetic determinants of macroscopic currents: insights from coupling and uncoupling of GABAA receptor desensitization and deactivation. J Physiol. 2007 Nov 01; 584(Pt 3):769-87.
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The efficacy and tolerability of tiagabine in adult patients with post-traumatic stress disorder. J Clin Psychopharmacol. 2007 Feb; 27(1):85-8.
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Identification of structures within GABAA receptor alpha subunits that regulate the agonist action of pentobarbital. J Pharmacol Exp Ther. 2006 Sep; 318(3):1094-101.
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LTP in the lateral amygdala during cocaine withdrawal. Eur J Neurosci. 2006 Jan; 23(1):239-50.