Scavenger Receptors, Class B
"Scavenger Receptors, Class B" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A family of scavenger receptors that are predominately localized to CAVEOLAE of the PLASMA MEMBRANE and bind HIGH DENSITY LIPOPROTEINS.
| Descriptor ID |
D051122
|
| MeSH Number(s) |
D12.776.543.750.705.940.625 D12.776.543.750.710.450.750.625
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Scavenger Receptors, Class B".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Membrane Proteins [D12.776.543]
- Receptors, Cell Surface [D12.776.543.750]
- Receptors, Immunologic [D12.776.543.750.705]
- Receptors, Scavenger [D12.776.543.750.705.940]
- Scavenger Receptors, Class B [D12.776.543.750.705.940.625]
- Receptors, Lipoprotein [D12.776.543.750.710]
- Receptors, LDL [D12.776.543.750.710.450]
- Receptors, Scavenger [D12.776.543.750.710.450.750]
- Scavenger Receptors, Class B [D12.776.543.750.710.450.750.625]
Below are MeSH descriptors whose meaning is more specific than "Scavenger Receptors, Class B".
This graph shows the total number of publications written about "Scavenger Receptors, Class B" by people in this website by year, and whether "Scavenger Receptors, Class B" was a major or minor topic of these publications.
To see the data from this visualization as text,
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2002 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2010 | 1 | 1 | 2 |
| 2011 | 0 | 1 | 1 |
| 2014 | 1 | 0 | 1 |
| 2015 | 0 | 1 | 1 |
| 2016 | 1 | 0 | 1 |
| 2018 | 1 | 0 | 1 |
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Below are the most recent publications written about "Scavenger Receptors, Class B" by people in Profiles.
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Protein markers of dysfunctional HDL in scavenger receptor class B type I deficient mice. J Transl Med. 2018 06 07; 16(1):155.
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S1P in HDL promotes interaction between SR-BI and S1PR1 and activates S1PR1-mediated biological functions: calcium flux and S1PR1 internalization. J Lipid Res. 2017 02; 58(2):325-338.
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A genetic dissection of intestinal fat-soluble vitamin and carotenoid absorption. Hum Mol Genet. 2015 Jun 01; 24(11):3206-19.
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Formation of a protein corona on silver nanoparticles mediates cellular toxicity via scavenger receptors. Toxicol Sci. 2015 Jan; 143(1):136-46.
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Lipid-free apolipoprotein A-I and discoidal reconstituted high-density lipoproteins differentially inhibit glucose-induced oxidative stress in human macrophages. Arterioscler Thromb Vasc Biol. 2011 May; 31(5):1192-200.
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ISX is a retinoic acid-sensitive gatekeeper that controls intestinal beta,beta-carotene absorption and vitamin A production. FASEB J. 2010 Jun; 24(6):1656-66.
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High-density lipoprotein metabolism and the human embryo. Hum Reprod Update. 2010 Jan-Feb; 16(1):20-38.
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Insulin-like growth factor binding protein-3 mediates vascular repair by enhancing nitric oxide generation. Circ Res. 2009 Oct 23; 105(9):897-905.
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Impaired secretion of apolipoprotein E2 from macrophages. J Biol Chem. 2007 May 04; 282(18):13746-53.
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Overexpression of SR-BI by adenoviral vector promotes clearance of apoA-I, but not apoB, in human apoB transgenic mice. J Lipid Res. 2002 Sep; 43(9):1421-8.