Home
About
Overview
Sharing Data
ORCID
Help
History (1)
GSNO attenuates EAE disease by S-nitrosylation-mediated modulation of endothelial-monocyte interactions.
See All Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
GSNO attenuates EAE disease by S-nitrosylation-mediated modulation of endothelial-monocyte interactions.
GSNO attenuates EAE disease by S-nitrosylation-mediated modulation of endothelial-monocyte interactions. Glia. 2007 Jan 01; 55(1):65-77.
View in:
PubMed
subject areas
Administration, Oral
Animals
Blood-Brain Barrier
Cell Adhesion
Cell Adhesion Molecules
Cell Line
Central Nervous System
Chemotaxis, Leukocyte
Disease Models, Animal
Encephalomyelitis, Autoimmune, Experimental
Endothelial Cells
Female
Mice
Monocytes
NF-kappa B
Nitric Oxide Donors
Nitro Compounds
Rats
Rats, Inbred Lew
Signal Transduction
S-Nitrosoglutathione
Transcription Factor RelA
Tumor Necrosis Factor-alpha
authors with profiles
Avtar K. Singh
Inderjit Singh