Home
About
Overview
Sharing Data
ORCID
Help
History (2)
AKT methylation by SETDB1 promotes AKT kinase activity and oncogenic functions.
Further development of a model of chronic bone marrow aplasia in the busulphan-treated mouse.
See All 2 Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
AKT methylation by SETDB1 promotes AKT kinase activity and oncogenic functions.
AKT methylation by SETDB1 promotes AKT kinase activity and oncogenic functions. Nat Cell Biol. 2019 02; 21(2):226-237.
View in:
PubMed
subject areas
Animals
Antibiotics, Antineoplastic
Carcinogenesis
Cell Line, Tumor
Female
HEK293 Cells
Histone-Lysine N-Methyltransferase
Humans
Methylation
Mice, Nude
Mice, Transgenic
Neoplasms
Plicamycin
Protein Methyltransferases
Proto-Oncogene Proteins c-akt
Sf9 Cells
Spodoptera
Xenograft Model Antitumor Assays
authors with profiles
Wenjian Gan