Epithelial-Mesenchymal Transition
"Epithelial-Mesenchymal Transition" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Phenotypic changes of EPITHELIAL CELLS to MESENCHYME type, which increase cell mobility critical in many developmental processes such as NEURAL TUBE development. NEOPLASM METASTASIS and DISEASE PROGRESSION may also induce this transition.
Descriptor ID |
D058750
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MeSH Number(s) |
G04.356.500
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Concept/Terms |
Epithelial-Mesenchymal Transition- Epithelial-Mesenchymal Transition
- Epithelial Mesenchymal Transition
- Epithelial-Mesenchymal Transitions
- Transition, Epithelial-Mesenchymal
- Transitions, Epithelial-Mesenchymal
- Epithelial-Mesenchymal Transformation
- Epithelial Mesenchymal Transformation
- Epithelial-Mesenchymal Transformations
- Transformation, Epithelial-Mesenchymal
- Transformations, Epithelial-Mesenchymal
|
Below are MeSH descriptors whose meaning is more general than "Epithelial-Mesenchymal Transition".
Below are MeSH descriptors whose meaning is more specific than "Epithelial-Mesenchymal Transition".
This graph shows the total number of publications written about "Epithelial-Mesenchymal Transition" by people in this website by year, and whether "Epithelial-Mesenchymal Transition" was a major or minor topic of these publications.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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2010 | 0 | 1 | 1 |
2011 | 5 | 3 | 8 |
2012 | 4 | 2 | 6 |
2013 | 5 | 2 | 7 |
2014 | 1 | 8 | 9 |
2015 | 5 | 2 | 7 |
2016 | 1 | 3 | 4 |
2017 | 2 | 2 | 4 |
2018 | 3 | 5 | 8 |
2019 | 1 | 3 | 4 |
2020 | 0 | 3 | 3 |
2021 | 1 | 6 | 7 |
2022 | 1 | 1 | 2 |
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Below are the most recent publications written about "Epithelial-Mesenchymal Transition" by people in Profiles.
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NNMT contributes to high metastasis of triple negative breast cancer by enhancing PP2A/MEK/ERK/c-Jun/ABCA1 pathway mediated membrane fluidity. Cancer Lett. 2022 10 28; 547:215884.
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The ubiquitin E3 ligase ARIH1 regulates hnRNP E1 protein stability, EMT and breast cancer progression. Oncogene. 2022 03; 41(12):1679-1690.
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TGF?-induced expression of long noncoding lincRNA Platr18 controls breast cancer axonogenesis. Life Sci Alliance. 2022 02; 5(2).
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Aortic valve disease in diabetes: Molecular mechanisms and novel therapies. J Cell Mol Med. 2021 10; 25(20):9483-9495.
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Autocrine signaling by receptor tyrosine kinases in urothelial carcinoma of the bladder. PLoS One. 2021; 16(7):e0241766.
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BOO induces fibrosis and EMT in urothelial cells which can be recapitulated in vitro through elevated storage and voiding pressure cycles. Int Urol Nephrol. 2021 Oct; 53(10):2007-2018.
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High-Fat Diet Drives an Aggressive Pancreatic Cancer Phenotype. J Surg Res. 2021 08; 264:163-172.
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Mevalonate Pathway Inhibition Slows Breast Cancer Metastasis via Reduced N-glycosylation Abundance and Branching. Cancer Res. 2021 05 15; 81(10):2625-2635.
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Low Expression of Rasal2 Promotes Non-small Cell Lung Cancer Metastasis through Ras/ERK Pathway. Biol Pharm Bull. 2021; 44(7):992-998.
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Emodin reduces Breast Cancer Lung Metastasis by suppressing Macrophage-induced Breast Cancer Cell Epithelial-mesenchymal transition and Cancer Stem Cell formation. Theranostics. 2020; 10(18):8365-8381.