Polychlorinated Dibenzodioxins
"Polychlorinated Dibenzodioxins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Dibenzodioxin derivatives that contain multiple chloride atoms bound to the benzene ring structures.
| Descriptor ID |
D000072317
|
| MeSH Number(s) |
D02.309.500.450 D03.633.300.786
|
| Concept/Terms |
Polychlorinated Dibenzodioxins- Polychlorinated Dibenzodioxins
- Dibenzodioxins, Polychlorinated
- Polychlorinated Dibenzo-p-dioxins
- Dibenzo-p-dioxins, Polychlorinated
- Polychlorinated Dibenzo p dioxins
- Chlorinated Dibenzo-p-dioxins
- Chlorinated Dibenzo p dioxins
- Dibenzo-p-dioxins, Chlorinated
- Polychlorinated Dibenzodioxin
- Dibenzodioxin, Polychlorinated
Polychlorodibenzo-4-dioxin- Polychlorodibenzo-4-dioxin
- Polychlorodibenzo 4 dioxin
- PCDD
- Polychlorodibenzo-p-dioxin
- Polychlorodibenzo p dioxin
2,3,7,8-Tetrachlorodibenzo-p-dioxin- 2,3,7,8-Tetrachlorodibenzo-p-dioxin
- TCDD
- Dibenzo(b,e)(1,4)dioxin, 2,3,7,8-tetrachloro-
|
Below are MeSH descriptors whose meaning is more general than "Polychlorinated Dibenzodioxins".
Below are MeSH descriptors whose meaning is more specific than "Polychlorinated Dibenzodioxins".
This graph shows the total number of publications written about "Polychlorinated Dibenzodioxins" by people in this website by year, and whether "Polychlorinated Dibenzodioxins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 1 | 0 | 1 |
| 1997 | 1 | 1 | 2 |
| 1998 | 3 | 0 | 3 |
| 1999 | 1 | 0 | 1 |
| 2001 | 1 | 0 | 1 |
| 2002 | 1 | 0 | 1 |
| 2003 | 1 | 0 | 1 |
| 2004 | 4 | 1 | 5 |
| 2005 | 3 | 0 | 3 |
| 2006 | 1 | 0 | 1 |
| 2008 | 1 | 0 | 1 |
| 2010 | 1 | 0 | 1 |
| 2011 | 0 | 1 | 1 |
| 2012 | 1 | 0 | 1 |
| 2015 | 1 | 0 | 1 |
| 2017 | 0 | 1 | 1 |
| 2018 | 0 | 1 | 1 |
| 2019 | 2 | 1 | 3 |
| 2020 | 2 | 0 | 2 |
| 2021 | 2 | 1 | 3 |
| 2022 | 1 | 0 | 1 |
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Below are the most recent publications written about "Polychlorinated Dibenzodioxins" by people in Profiles.
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AhR Activation Leads to Attenuation of Murine Autoimmune Hepatitis: Single-Cell RNA-Seq Analysis Reveals Unique Immune Cell Phenotypes and Gene Expression Changes in the Liver. Front Immunol. 2022; 13:899609.
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Effects of Acute 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Exposure on the Circulating and Cecal Metabolome Profile. Int J Mol Sci. 2021 Oct 30; 22(21).
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The Ability of AhR Ligands to Attenuate Delayed Type Hypersensitivity Reaction Is Associated With Alterations in the Gut Microbiota. Front Immunol. 2021; 12:684727.
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AhR Ligands Differentially Regulate miRNA-132 Which Targets HMGB1 and to Control the Differentiation of Tregs and Th-17 Cells During Delayed-Type Hypersensitivity Response. Front Immunol. 2021; 12:635903.
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AhR Activation Leads to Alterations in the Gut Microbiome with Consequent Effect on Induction of Myeloid Derived Suppressor Cells in a CXCR2-Dependent Manner. Int J Mol Sci. 2020 Dec 17; 21(24).
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From Suppressor T cells to Regulatory T cells: How the Journey That Began with the Discovery of the Toxic Effects of TCDD Led to Better Understanding of the Role of AhR in Immunoregulation. Int J Mol Sci. 2020 Oct 22; 21(21).
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AhR Activation Leads to Massive Mobilization of Myeloid-Derived Suppressor Cells with Immunosuppressive Activity through Regulation of CXCR2 and MicroRNA miR-150-5p and miR-543-3p That Target Anti-Inflammatory Genes. J Immunol. 2019 10 01; 203(7):1830-1844.
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Computational prediction and in vitro validation of VEGFR1 as a novel protein target for 2,3,7,8-tetrachlorodibenzo-p-dioxin. Sci Rep. 2019 05 02; 9(1):6810.
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The impact of maternally derived dioxins on embryonic development and hepatic AHR signaling in a long-lived apex predator. Chemosphere. 2019 Aug; 229:489-499.
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Comparative analysis of interactions between aryl hydrocarbon receptor ligand binding domain with its ligands: a computational study. BMC Struct Biol. 2018 12 06; 18(1):15.