Mitochondrial Proton-Translocating ATPases
"Mitochondrial Proton-Translocating ATPases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proton-translocating ATPases responsible for ADENOSINE TRIPHOSPHATE synthesis in the MITOCHONDRIA. They derive energy from the respiratory chain-driven reactions that develop high concentrations of protons within the intermembranous space of the mitochondria.
| Descriptor ID |
D025261
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| MeSH Number(s) |
D08.811.277.040.025.325.750 D08.811.913.696.650.150.500.750 D12.776.157.530.450.250.875.500.750 D12.776.543.585.450.250.875.500.750 D12.776.543.585.475.625 D12.776.575.750.625
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| Concept/Terms |
Mitochondrial Proton-Translocating ATPases- Mitochondrial Proton-Translocating ATPases
- ATPases, Mitochondrial Proton-Translocating
- Mitochondrial Proton Translocating ATPases
- Proton-Translocating ATPases, Mitochondrial
- Mitochondrial F(1)F(0) ATPase
- Mitochondrial ATP Synthase
- ATP Synthase, Mitochondrial
- Mitochondrial ATP Synthases
- ATP Synthases, Mitochondrial
|
Below are MeSH descriptors whose meaning is more general than "Mitochondrial Proton-Translocating ATPases".
Below are MeSH descriptors whose meaning is more specific than "Mitochondrial Proton-Translocating ATPases".
This graph shows the total number of publications written about "Mitochondrial Proton-Translocating ATPases" by people in this website by year, and whether "Mitochondrial Proton-Translocating ATPases" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2012 | 1 | 1 | 2 |
| 2013 | 0 | 2 | 2 |
| 2015 | 1 | 0 | 1 |
| 2016 | 0 | 1 | 1 |
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Below are the most recent publications written about "Mitochondrial Proton-Translocating ATPases" by people in Profiles.
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Genome-Wide Association Study in an Amerindian Ancestry Population Reveals Novel Systemic Lupus Erythematosus Risk Loci and the Role of European Admixture. Arthritis Rheumatol. 2016 Apr; 68(4):932-43.
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Urinary ATP Synthase Subunit ? Is a Novel Biomarker of Renal Mitochondrial Dysfunction in Acute Kidney Injury. Toxicol Sci. 2015 May; 145(1):108-17.
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Diggin' on u(biquitin): a novel method for the identification of physiological E3 ubiquitin ligase substrates. Cell Biochem Biophys. 2013 Sep; 67(1):127-38.
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Green tea polyphenols stimulate mitochondrial biogenesis and improve renal function after chronic cyclosporin a treatment in rats. PLoS One. 2014; 8(6):e65029.
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Genetic dysfunction of MT-ATP6 causes axonal Charcot-Marie-Tooth disease. Neurology. 2012 Sep 11; 79(11):1145-54.
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Calpain 10 homology modeling with CYGAK and increased lipophilicity leads to greater potency and efficacy in cells. ACS Chem Biol. 2012 Aug 17; 7(8):1410-9.
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Contribution of impaired myocardial insulin signaling to mitochondrial dysfunction and oxidative stress in the heart. Circulation. 2009 Mar 10; 119(9):1272-83.
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Long-chain ceramide is a potent inhibitor of the mitochondrial permeability transition pore. J Biol Chem. 2008 Sep 05; 283(36):24707-17.
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The prolyl hydroxylase oxygen-sensing pathway is cytoprotective and allows maintenance of mitochondrial membrane potential during metabolic inhibition. Am J Physiol Cell Physiol. 2007 Feb; 292(2):C719-28.