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    • Bortezomib reverses a post-translational mechanism of tumorigenesis for patched1 haploinsufficiency in medulloblastoma.
    • Changes in the structural composition and reactivity of Acer rubrum leaf litter tannins exposed to warming and altered precipitation: climatic stress-induced tannins are more reactive.
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Bortezomib reverses a post-translational mechanism of tumorigenesis for patched1 haploinsufficiency in medulloblastoma.

Bortezomib reverses a post-translational mechanism of tumorigenesis for patched1 haploinsufficiency in medulloblastoma. Pediatr Blood Cancer. 2009 Aug; 53(2):136-44.

View in: PubMed

subject areas
  • Animals
  • Antineoplastic Agents
  • Boronic Acids
  • Bortezomib
  • Cell Line, Tumor
  • Cell Transformation, Neoplastic
  • Cerebellar Neoplasms
  • Disease Models, Animal
  • Gene Knock-In Techniques
  • Hedgehog Proteins
  • Humans
  • Immunoblotting
  • Immunoprecipitation
  • Loss of Heterozygosity
  • Medulloblastoma
  • Mice
  • Mice, Knockout
  • Patched Receptors
  • Patched-1 Receptor
  • PAX7 Transcription Factor
  • Proteasome Endopeptidase Complex
  • Protein Isoforms
  • Protein Processing, Post-Translational
  • Pyrazines
  • Receptors, Cell Surface

authors with profiles
  • Denis C Guttridge