Nitric Oxide Synthase Type III
"Nitric Oxide Synthase Type III" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A CALCIUM-dependent, constitutively-expressed form of nitric oxide synthase found primarily in ENDOTHELIAL CELLS.
Descriptor ID |
D052250
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MeSH Number(s) |
D08.811.682.664.500.772.750
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Concept/Terms |
Nitric Oxide Synthase Type III- Nitric Oxide Synthase Type III
- Nitric Oxide Synthase, Type III
- Endothelial Nitric Oxide Synthase
- ENOS Enzyme
- ECNOS Enzyme
- Endothelial Constitutive Nitric Oxide Synthase
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Below are MeSH descriptors whose meaning is more general than "Nitric Oxide Synthase Type III".
Below are MeSH descriptors whose meaning is more specific than "Nitric Oxide Synthase Type III".
This graph shows the total number of publications written about "Nitric Oxide Synthase Type III" by people in this website by year, and whether "Nitric Oxide Synthase Type III" was a major or minor topic of these publications.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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1998 | 0 | 1 | 1 |
2000 | 0 | 1 | 1 |
2003 | 0 | 1 | 1 |
2004 | 0 | 2 | 2 |
2005 | 1 | 4 | 5 |
2006 | 1 | 1 | 2 |
2007 | 3 | 2 | 5 |
2008 | 1 | 1 | 2 |
2009 | 2 | 1 | 3 |
2010 | 4 | 1 | 5 |
2012 | 3 | 1 | 4 |
2013 | 5 | 4 | 9 |
2014 | 4 | 3 | 7 |
2015 | 2 | 1 | 3 |
2016 | 1 | 0 | 1 |
2017 | 2 | 3 | 5 |
2018 | 1 | 1 | 2 |
2019 | 1 | 0 | 1 |
2020 | 1 | 0 | 1 |
2021 | 1 | 0 | 1 |
2022 | 1 | 0 | 1 |
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Below are the most recent publications written about "Nitric Oxide Synthase Type III" by people in Profiles.
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?-PIX cooperates with GIT1 to regulate endothelial nitric oxide synthase in sinusoidal endothelial cells. Am J Physiol Gastrointest Liver Physiol. 2022 Nov 01; 323(5):G511-G522.
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Endothelial Nitric Oxide Synthase-Deficient Mice: A Model of Spontaneous Cerebral Small-Vessel Disease. Am J Pathol. 2021 11; 191(11):1932-1945.
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?-Arrestin2 is a critical component of the GPCR-eNOS signalosome. Proc Natl Acad Sci U S A. 2020 05 26; 117(21):11483-11492.
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S-Nitrosoglutathione Mimics the Beneficial Activity of Endothelial Nitric Oxide Synthase-Derived Nitric Oxide in a Mouse Model of Stroke. J Stroke Cerebrovasc Dis. 2019 Dec; 28(12):104470.
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Novel Potentials of the DPP-4 Inhibitor Sitagliptin against Ischemia-Reperfusion (I/R) Injury in Rat Ex-Vivo Heart Model. Int J Mol Sci. 2018 Oct 18; 19(10).
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Kallistatin attenuates endothelial senescence by modulating Let-7g-mediated miR-34a-SIRT1-eNOS pathway. J Cell Mol Med. 2018 09; 22(9):4387-4398.
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Opposing Effects of Oxygen Regulation on Kallistatin Expression: Kallistatin as a Novel Mediator of Oxygen-Induced HIF-1-eNOS-NO Pathway. Oxid Med Cell Longev. 2017; 2017:5262958.
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Mechanisms of acute neurovascular protection with AT1 blockade after stroke: Effect of prestroke hypertension. PLoS One. 2017; 12(6):e0178867.
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Kallistatin reduces vascular senescence and aging by regulating microRNA-34a-SIRT1 pathway. Aging Cell. 2017 08; 16(4):837-846.
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Caveolin 1 and G-Protein-Coupled Receptor Kinase-2 Coregulate Endothelial Nitric Oxide Synthase Activity in Sinusoidal Endothelial Cells. Am J Pathol. 2017 Apr; 187(4):896-907.