"Rats, Inbred Dahl" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Inbred rats derived from Sprague-Dawley rats and used for the study of salt-dependent hypertension. Salt-sensitive and salt-resistant strains have been selectively bred to show the opposite genetically determined blood pressure responses to excess sodium chloride ingestion.
| Descriptor ID |
D020303
|
| MeSH Number(s) |
B01.050.050.199.520.760.165 B01.050.150.900.649.313.992.635.505.700.400.165
|
| Concept/Terms |
Rats, Inbred Dahl- Rats, Inbred Dahl
- Dahl Rats, Inbred
- Inbred Dahl Rats
- Dahl Rats
- Rats, Dahl
Dahl Salt-Resistant Rats- Dahl Salt-Resistant Rats
- Dahl Salt Resistant Rats
- Rats, Dahl Salt-Resistant
- Salt-Resistant Rats, Dahl
Dahl Salt-Sensitive Rats- Dahl Salt-Sensitive Rats
- Dahl Salt Sensitive Rats
- Rats, Dahl Salt-Sensitive
- Salt-Sensitive Rats, Dahl
- Dahl Hypertensive Rats
- Hypertensive Rats, Dahl
- Rats, Dahl Hypertensive
|
Below are MeSH descriptors whose meaning is more general than "Rats, Inbred Dahl".
Below are MeSH descriptors whose meaning is more specific than "Rats, Inbred Dahl".
This graph shows the total number of publications written about "Rats, Inbred Dahl" by people in this website by year, and whether "Rats, Inbred Dahl" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 1999 | 0 | 2 | 2 |
| 2000 | 0 | 2 | 2 |
| 2001 | 0 | 2 | 2 |
| 2002 | 0 | 2 | 2 |
| 2003 | 1 | 1 | 2 |
| 2004 | 0 | 2 | 2 |
| 2006 | 0 | 2 | 2 |
| 2007 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2013 | 0 | 2 | 2 |
| 2015 | 0 | 1 | 1 |
| 2016 | 0 | 2 | 2 |
| 2017 | 0 | 2 | 2 |
| 2018 | 0 | 2 | 2 |
| 2019 | 0 | 1 | 1 |
| 2020 | 0 | 3 | 3 |
| 2021 | 0 | 1 | 1 |
| 2022 | 0 | 1 | 1 |
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Below are the most recent publications written about "Rats, Inbred Dahl" by people in Profiles.
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Effects of elevation of ANP and its deficiency on cardiorenal function. JCI Insight. 2022 05 09; 7(9).
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Inhibition of neprilysin with sacubitril without RAS blockage aggravates renal disease in Dahl SS rats. Ren Fail. 2021 Dec; 43(1):315-324.
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NOX4-dependent regulation of ENaC in hypertension and diabetic kidney disease. FASEB J. 2020 10; 34(10):13396-13408.
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Differential effects of low-dose sacubitril and/or valsartan on renal disease in salt-sensitive hypertension. Am J Physiol Renal Physiol. 2020 07 01; 319(1):F63-F75.
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Comprehensive assessment of mitochondrial respiratory function in freshly isolated nephron segments. Am J Physiol Renal Physiol. 2020 05 01; 318(5):F1237-F1245.
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Neurovascular protection in voltage-gated proton channel Hv1 knock-out rats after ischemic stroke: interaction with Na+ /H+ exchanger-1 antagonism. Physiol Rep. 2019 08; 7(13):e14142.
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Protective role of Trpc6 knockout in the progression of diabetic kidney disease. Am J Physiol Renal Physiol. 2018 10 01; 315(4):F1091-F1097.
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A NOX4/TRPC6 Pathway in Podocyte Calcium Regulation and Renal Damage in Diabetic Kidney Disease. J Am Soc Nephrol. 2018 07; 29(7):1917-1927.
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Nitric oxide production by glomerular podocytes. Nitric Oxide. 2018 01 30; 72:24-31.
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Essential role of Kir5.1 channels in renal salt handling and blood pressure control. JCI Insight. 2017 09 21; 2(18).