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12(S)-HETE increases the motility of prostate tumor cells through selective activation of PKC alpha.
Absence of Nrf2 or its selective overexpression in neurons and muscle does not affect survival in ALS-linked mutant hSOD1 mouse models.
The Digital Slide Archive: A Software Platform for Management, Integration, and Analysis of Histology for Cancer Research.
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12(S)-HETE increases the motility of prostate tumor cells through selective activation of PKC alpha.
12(S)-HETE increases the motility of prostate tumor cells through selective activation of PKC alpha. Adv Exp Med Biol. 1997; 400B:707-18.
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PubMed
subject areas
12-Hydroxy-5,8,10,14-eicosatetraenoic Acid
Adenocarcinoma
Animals
Calcium
Cell Movement
Chelating Agents
Egtazic Acid
Enzyme Activation
Enzyme Inhibitors
Humans
Isoenzymes
Male
Naphthalenes
Prostatic Neoplasms
Protein Kinase C
Protein Kinase C-alpha
Protein Kinase C-delta
Rats
Tumor Cells, Cultured
authors with profiles
Yusuf A. Hannun