3,3'-diindolylmethane ameliorates experimental autoimmune encephalomyelitis by promoting cell cycle arrest and apoptosis in activated T cells through microRNA signaling pathways.

3,3'-diindolylmethane ameliorates experimental autoimmune encephalomyelitis by promoting cell cycle arrest and apoptosis in activated T cells through microRNA signaling pathways. J Pharmacol Exp Ther. 2014 Aug; 350(2):341-52.

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subject areas

Animals
Apoptosis
Cell Cycle Checkpoints
Cells, Cultured
Core Binding Factor Alpha 2 Subunit
Encephalomyelitis, Autoimmune, Experimental
Female
Indoles
Lymphocyte Activation
Mice
Mice, Inbred C57BL
MicroRNAs
Proto-Oncogene Proteins c-bcl-2
Signal Transduction
T-Lymphocytes

authors with profiles

Mitzi Nagarkatti