Bone marrow transplantation reproduces the tristetraprolin-deficiency syndrome in recombination activating gene-2 (-/-) mice. Evidence that monocyte/macrophage progenitors may be responsible for TNFalpha overproduction.

Bone marrow transplantation reproduces the tristetraprolin-deficiency syndrome in recombination activating gene-2 (-/-) mice. Evidence that monocyte/macrophage progenitors may be responsible for TNFalpha overproduction. J Clin Invest. 1997 Sep 01; 100(5):986-95.

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subject areas

Animals
Autoantibodies
Body Weight
Bone Marrow Transplantation
DNA-Binding Proteins
Female
Immediate-Early Proteins
In Situ Hybridization
Macrophages
Male
Mice
Monocytes
Proteins
Stem Cells
Syndrome
Tristetraprolin
Tumor Necrosis Factor-alpha
Y Chromosome

authors with profiles

Gary S. Gilkeson