Polycystic Kidney Diseases
"Polycystic Kidney Diseases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Hereditary diseases that are characterized by the progressive expansion of a large number of tightly packed CYSTS within the KIDNEYS. They include diseases with autosomal dominant and autosomal recessive inheritance.
| Descriptor ID |
D007690
|
| MeSH Number(s) |
C12.777.419.403.875 C13.351.968.419.403.875 C16.131.077.717 C16.320.798
|
| Concept/Terms |
Polycystic Kidney Diseases- Polycystic Kidney Diseases
- Disease, Polycystic Kidney
- Diseases, Polycystic Kidney
- Kidney Disease, Polycystic
- Kidney Diseases, Polycystic
- Polycystic Kidney Disease
- Polycystic Renal Disease
- Disease, Polycystic Renal
- Diseases, Polycystic Renal
- Polycystic Renal Diseases
- Renal Disease, Polycystic
- Renal Diseases, Polycystic
Kidney, Polycystic- Kidney, Polycystic
- Kidneys, Polycystic
- Polycystic Kidneys
- Polycystic Kidney
|
Below are MeSH descriptors whose meaning is more general than "Polycystic Kidney Diseases".
Below are MeSH descriptors whose meaning is more specific than "Polycystic Kidney Diseases".
This graph shows the total number of publications written about "Polycystic Kidney Diseases" by people in this website by year, and whether "Polycystic Kidney Diseases" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 1 | 0 | 1 |
| 2002 | 1 | 0 | 1 |
| 2003 | 1 | 0 | 1 |
| 2009 | 1 | 0 | 1 |
| 2010 | 1 | 0 | 1 |
| 2011 | 2 | 0 | 2 |
| 2014 | 3 | 0 | 3 |
| 2015 | 3 | 0 | 3 |
| 2016 | 1 | 0 | 1 |
| 2019 | 1 | 0 | 1 |
| 2020 | 1 | 1 | 2 |
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Below are the most recent publications written about "Polycystic Kidney Diseases" by people in Profiles.
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Interferon Regulatory Factor-5 in Resident Macrophage Promotes Polycystic Kidney Disease. Kidney360. 2020 Mar; 1(3):179-190.
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Defining the human kidney N-glycome in normal and cancer tissues using MALDI imaging mass spectrometry. J Mass Spectrom. 2020 Apr; 55(4):e4490.
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Loss of primary cilia increases polycystin-2 and TRPV4 and the appearance of a nonselective cation channel in the mouse cortical collecting duct. Am J Physiol Renal Physiol. 2019 09 01; 317(3):F632-F637.
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Functional and therapeutic importance of purinergic signaling in polycystic kidney disease. Am J Physiol Renal Physiol. 2016 12 01; 311(6):F1135-F1139.
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Suppressing angiotensinogen synthesis attenuates kidney cyst formation in a Pkd1 mouse model. FASEB J. 2016 Jan; 30(1):370-9.
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The cpk model of recessive PKD shows glutamine dependence associated with the production of the oncometabolite 2-hydroxyglutarate. Am J Physiol Renal Physiol. 2015 Sep 15; 309(6):F492-8.
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Hyperglycemia in the absence of cilia accelerates cystogenesis and induces renal damage. Am J Physiol Renal Physiol. 2015 Jul 01; 309(1):F79-87.
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A possible zebrafish model of polycystic kidney disease: knockdown of wnt5a causes cysts in zebrafish kidneys. J Vis Exp. 2014 Dec 02; (94).
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Epidermal growth factor-induced proliferation of collecting duct cells from Oak Ridge polycystic kidney mice involves activation of Na+/H+ exchanger. Am J Physiol Cell Physiol. 2014 Sep 15; 307(6):C554-60.
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ADAM17 promotes proliferation of collecting duct kidney epithelial cells through ERK activation and increased glycolysis in polycystic kidney disease. Am J Physiol Renal Physiol. 2014 Sep 01; 307(5):F551-9.